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Sabita Roy

Department of Surgery

University of Minnesota

Minneapolis

MN, 55455

USA

[email]@umn.edu

Name/email consistency: medium

 
 
 
 
 
 
 

Affiliations

  • Department of Surgery, University of Minnesota, Minneapolis, MN, 55455, USA. 2005 - 2011
  • Department of Surgery, University of Minnesota, Minneapolis Veterans Affairs Medical Center, Minneapolis, USA. 2004 - 2005

References

  1. Opioid drug abuse and modulation of immune function: consequences in the susceptibility to opportunistic infections. Roy, S., Ninkovic, J., Banerjee, S., Charboneau, R.G., Das, S., Dutta, R., Kirchner, V.A., Koodie, L., Ma, J., Meng, J., Barke, R.A. J. Neuroimmune. Pharmacol (2011) [Pubmed]
  2. Modulation of immune function by morphine: implications for susceptibility to infection. Roy, S., Wang, J., Kelschenbach, J., Koodie, L., Martin, J. J. Neuroimmune. Pharmacol (2006) [Pubmed]
  3. In vivo activation of a mutant mu-opioid receptor by naltrexone produces a potent analgesic effect but no tolerance: role of mu-receptor activation and delta-receptor blockade in morphine tolerance. Roy, S., Guo, X., Kelschenbach, J., Liu, Y., Loh, H.H. J. Neurosci. (2005) [Pubmed]
  4. Morphine induces CD4+ T cell IL-4 expression through an adenylyl cyclase mechanism independent of the protein kinase A pathway. Roy, S., Wang, J., Charboneau, R., Loh, H.H., Barke, R.A. J. Immunol. (2005) [Pubmed]
  5. Chronic morphine treatment differentiates T helper cells to Th2 effector cells by modulating transcription factors GATA 3 and T-bet. Roy, S., Wang, J., Gupta, S., Charboneau, R., Loh, H.H., Barke, R.A. J. Neuroimmunol. (2004) [Pubmed]
 
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