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R. Paul Robertson

Division of Endocrinology and Metabolism

Pacific Northwest Diabetes Research Institute

University of Washington

720 Broadway

USA

[email]@pnri.org

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Division of Endocrinology and Metabolism, Pacific Northwest Diabetes Research Institute, University of Washington, 720 Broadway, USA. 2010
  • Department of Medicine, University of Washington, 1959 N.E. Pacific Street, Seattle, USA. 2010
  • Pacific Northwest Research Institute, 720 Broadway, Seattle, WA 98122, USA. 2004 - 2009
  • Department of Medicine and Department of Pharmacology, Pacific Northwest Research Institute, 720 Broadway, Seattle, USA. 2006
  • Pacific Northwest Research Institute and the Division of Metabolism, Endocrinology, and Nutrition, Department of Medicine, USA. 2004

References

  1. Update on transplanting beta cells for reversing type 1 diabetes. Robertson, R.P. Endocrinol. Metab. Clin. North Am. (2010) [Pubmed]
  2. Antioxidant drugs for treating beta-cell oxidative stress in type 2 diabetes: glucose-centric versus insulin-centric therapy. Robertson, R.P. Discov. Med (2010) [Pubmed]
  3. Islet transplantation a decade later and strategies for filling a half-full glass. Robertson, R.P. Diabetes (2010) [Pubmed]
  4. 2009 presidential address: mentoring ... touching the future. Robertson, R.P. Diabetes. Care (2009) [Pubmed]
  5. Estimation of beta-cell mass by metabolic tests: necessary, but how sufficient? Robertson, R.P. Diabetes (2007) [Pubmed]
  6. Chronic oxidative stress as a mechanism for glucose toxicity of the beta cell in type 2 diabetes. Robertson, R., Zhou, H., Zhang, T., Harmon, J.S. Cell Biochem. Biophys. (2007) [Pubmed]
  7. Pancreatic islet beta-cell and oxidative stress: the importance of glutathione peroxidase. Robertson, R.P., Harmon, J.S. FEBS Lett. (2007) [Pubmed]
  8. Oxidative stress and impaired insulin secretion in type 2 diabetes. Robertson, R.P. Curr. Opin. Pharmacol (2006) [Pubmed]
  9. Diabetes, glucose toxicity, and oxidative stress: A case of double jeopardy for the pancreatic islet beta cell. Robertson, R.P., Harmon, J.S. Free Radic. Biol. Med. (2006) [Pubmed]
  10. Prevention of oxidative stress by adenoviral overexpression of glutathione-related enzymes in pancreatic islets. Robertson, R.P., Tanaka, Y., Takahashi, H., Tran, P.O., Harmon, J.S. Ann. N. Y. Acad. Sci. (2005) [Pubmed]
  11. Islet transplantation as a treatment for diabetes - a work in progress. Robertson, R.P. N. Engl. J. Med. (2004) [Pubmed]
  12. Consequences on beta-cell function and reserve after long-term pancreas transplantation. Robertson, R.P. Diabetes (2004) [Pubmed]
  13. AIRarg and AIRgluc as predictors of insulin secretory reserve. Robertson, R.P. Transplant. Proc. (2004) [Pubmed]
  14. Chronic oxidative stress as a central mechanism for glucose toxicity in pancreatic islet beta cells in diabetes. Robertson, R.P. J. Biol. Chem. (2004) [Pubmed]
 
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