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Etsu Suzuki

Second Department of Internal Medicine

Faculty of Medicine

University of Tokyo

Tokyo

Japan

[email]@h.u-tokyo.ac.jp

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Second Department of Internal Medicine, Faculty of Medicine, University of Tokyo, Tokyo, Japan. 1999 - 2004

References

  1. Myocyte enhancer factor 2 mediates vascular inflammation via the p38-dependent pathway. Suzuki, E., Satonaka, H., Nishimatsu, H., Oba, S., Takeda, R., Omata, M., Fujita, T., Nagai, R., Hirata, Y. Circ. Res. (2004) [Pubmed]
  2. Angiotensin II induces myocyte enhancer factor 2- and calcineurin/nuclear factor of activated T cell-dependent transcriptional activation in vascular myocytes. Suzuki, E., Nishimatsu, H., Satonaka, H., Walsh, K., Goto, A., Omata, M., Fujita, T., Nagai, R., Hirata, Y. Circ. Res. (2002) [Pubmed]
  3. Constitutive activation of proto-oncogen protein p21 induces cell cycle arrest in the G1 phase in contact-inhibited vascular endothelial cells. Suzuki, E., Nishimatsu, H., Nagata, D., Satonaka, H., Goto, A., Omata, M., Fujita, T., Nagai, R., Hirata, Y. Hypertens. Res. (2002) [Pubmed]
  4. Reentry into the cell cycle of contact-inhibited vascular endothelial cells by a phosphatase inhibitor. Possible involvement of extracellular signal-regulated kinase and phosphatidylinositol 3-kinase. Suzuki, E., Nagata, D., Yoshizumi, M., Kakoki, M., Goto, A., Omata, M., Hirata, Y. J. Biol. Chem. (2000) [Pubmed]
  5. Molecular mechanisms of endothelin-1-induced cell-cycle progression: involvement of extracellular signal-regulated kinase, protein kinase C, and phosphatidylinositol 3-kinase at distinct points. Suzuki, E., Nagata, D., Kakoki, M., Hayakawa, H., Goto, A., Omata, M., Hirata, Y. Circ. Res. (1999) [Pubmed]
 
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