Wilhelm Kriz
Anatomy and Cell Biology
Medical Faculty Mannheim
University of Heidelberg
Heidelberg
Germany
Name/email consistency: high
- Ontogenetic development of the filtration barrier. Kriz, W. Nephron Exp. Nephrol. (2007)
- Pathways to nephron loss starting from glomerular diseases-insights from animal models. Kriz, W., LeHir, M. Kidney Int. (2005)
- TRPC6 - a new podocyte gene involved in focal segmental glomerulosclerosis. Kriz, W. Trends. Mol. Med (2005)
- Adenosine and ATP: traffic regulators in the kidney. Kriz, W. J. Clin. Invest. (2004)
- Pathways to recovery and loss of nephrons in anti-Thy-1 nephritis. Kriz, W., Hähnel, B., Hosser, H., Ostendorf, T., Gaertner, S., Kränzlin, B., Gretz, N., Shimizu, F., Floege, J. J. Am. Soc. Nephrol. (2003)
- Podocyte is the major culprit accounting for the progression of chronic renal disease. Kriz, W. Microsc. Res. Tech. (2002)
- Tracer studies in the rat demonstrate misdirected filtration and peritubular filtrate spreading in nephrons with segmental glomerulosclerosis. Kriz, W., Hartmann, I., Hosser, H., Hähnel, B., Kränzlin, B., Provoost, A., Gretz, N. J. Am. Soc. Nephrol. (2001)
- How does podocyte damage result in tubular damage?. Kriz, W., Elger, M., Hosser, H., Hähnel, B., Provoost, A., Kränzlin, B., Gretz, N. Kidney Blood Press. Res. (1999)
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