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André Báfica

Immunobiology Section

Laboratory of Parasitic Diseases

National Institute of Allergy and Infectious Diseases

National Institutes of Health

USA

[email]@niaid.nih.gov

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Immunobiology Section, Laboratory of Parasitic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, USA. 2004 - 2007
  • Immunobiology Section, National Institute of Allergy and Infectious Diseases and. Chemical Immunology Section, Laboratory of Cell Biology, National Cancer Institute, USA. 2003

References

  1. The IFN-inducible GTPase LRG47 (Irgm1) negatively regulates TLR4-triggered proinflammatory cytokine production and prevents endotoxemia. Bafica, A., Feng, C.G., Santiago, H.C., Aliberti, J., Cheever, A., Thomas, K.E., Taylor, G.A., Vogel, S.N., Sher, A. J. Immunol. (2007) [Pubmed]
  2. TLR9 regulates Th1 responses and cooperates with TLR2 in mediating optimal resistance to Mycobacterium tuberculosis. Bafica, A., Scanga, C.A., Feng, C.G., Leifer, C., Cheever, A., Sher, A. J. Exp. Med. (2005) [Pubmed]
  3. The induction of Toll-like receptor tolerance enhances rather than suppresses HIV-1 gene expression in transgenic mice. Báfica, A., Scanga, C.A., Equils, O., Sher, A. J. Leukoc. Biol. (2004) [Pubmed]
  4. Influence of coinfecting pathogens on HIV expression: evidence for a role of Toll-like receptors. Báfica, A., Scanga, C.A., Schito, M., Chaussabel, D., Sher, A. J. Immunol. (2004) [Pubmed]
  5. Cutting edge: in vivo induction of integrated HIV-1 expression by mycobacteria is critically dependent on Toll-like receptor 2. Báfica, A., Scanga, C.A., Schito, M.L., Hieny, S., Sher, A. J. Immunol. (2003) [Pubmed]
 
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