The world's first wiki where authorship really matters (Nature Genetics, 2008). Due credit and reputation for authors. Imagine a global collaborative knowledge base for original thoughts. Search thousands of articles and collaborate with scientists around the globe.

wikigene or wiki gene protein drug chemical gene disease author authorship tracking collaborative publishing evolutionary knowledge reputation system wiki2.0 global collaboration genes proteins drugs chemicals diseases compound
Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Bysani Chandrasekar

Department of Veterans Affairs South Texas Veterans Health Care System

San Antonio

Texas 78229-4404

USA

[email]@uthscsa.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Veterans Affairs South Texas Veterans Health Care System, San Antonio, Texas 78229-4404, USA. 2006 - 2008
  • Division of Cardiology, University of Texas Health Science Center, San Antonio, 78229-3900, USA. 2001 - 2006
  • Department of Medicine, University of Texas Health Science Center at San Antonio, South Texas Veterans Healthcare System, Audie Murphy Division, USA. 2001

References

  1. Interleukin-18 suppresses adiponectin expression in 3T3-L1 adipocytes via a novel signal transduction pathway involving ERK1/2-dependent NFATc4 phosphorylation. Chandrasekar, B., Patel, D.N., Mummidi, S., Kim, J.W., Clark, R.A., Valente, A.J. J. Biol. Chem. (2008) [Pubmed]
  2. Adiponectin blocks interleukin-18-mediated endothelial cell death via APPL1-dependent AMP-activated protein kinase (AMPK) activation and IKK/NF-kappaB/PTEN suppression. Chandrasekar, B., Boylston, W.H., Venkatachalam, K., Webster, N.J., Prabhu, S.D., Valente, A.J. J. Biol. Chem. (2008) [Pubmed]
  3. Interleukin-18 induces human cardiac endothelial cell death via a novel signaling pathway involving NF-kappaB-dependent PTEN activation. Chandrasekar, B., Valente, A.J., Freeman, G.L., Mahimainathan, L., Mummidi, S. Biochem. Biophys. Res. Commun. (2006) [Pubmed]
  4. Interleukin-18-induced human coronary artery smooth muscle cell migration is dependent on NF-kappaB- and AP-1-mediated matrix metalloproteinase-9 expression and is inhibited by atorvastatin. Chandrasekar, B., Mummidi, S., Mahimainathan, L., Patel, D.N., Bailey, S.R., Imam, S.Z., Greene, W.C., Valente, A.J. J. Biol. Chem. (2006) [Pubmed]
  5. The pro-atherogenic cytokine interleukin-18 induces CXCL16 expression in rat aortic smooth muscle cells via MyD88, interleukin-1 receptor-associated kinase, tumor necrosis factor receptor-associated factor 6, c-Src, phosphatidylinositol 3-kinase, Akt, c-Jun N-terminal kinase, and activator protein-1 signaling. Chandrasekar, B., Mummidi, S., Valente, A.J., Patel, D.N., Bailey, S.R., Freeman, G.L., Hatano, M., Tokuhisa, T., Jensen, L.E. J. Biol. Chem. (2005) [Pubmed]
  6. CXCL16 signals via Gi, phosphatidylinositol 3-kinase, Akt, I kappa B kinase, and nuclear factor-kappa B and induces cell-cell adhesion and aortic smooth muscle cell proliferation. Chandrasekar, B., Bysani, S., Mummidi, S. J. Biol. Chem. (2004) [Pubmed]
  7. Activation of intrinsic and extrinsic proapoptotic signaling pathways in interleukin-18-mediated human cardiac endothelial cell death. Chandrasekar, B., Vemula, K., Surabhi, R.M., Li-Weber, M., Owen-Schaub, L.B., Jensen, L.E., Mummidi, S. J. Biol. Chem. (2004) [Pubmed]
  8. Beta-adrenergic stimulation induces interleukin-18 expression via beta2-AR, PI3K, Akt, IKK, and NF-kappaB. Chandrasekar, B., Marelli-Berg, F.M., Tone, M., Bysani, S., Prabhu, S.D., Murray, D.R. Biochem. Biophys. Res. Commun. (2004) [Pubmed]
  9. Chemokine-cytokine cross-talk. The ELR+ CXC chemokine LIX (CXCL5) amplifies a proinflammatory cytokine response via a phosphatidylinositol 3-kinase-NF-kappa B pathway. Chandrasekar, B., Melby, P.C., Sarau, H.M., Raveendran, M., Perla, R.P., Marelli-Berg, F.M., Dulin, N.O., Singh, I.S. J. Biol. Chem. (2003) [Pubmed]
  10. TNF-alpha and H2O2 induce IL-18 and IL-18R beta expression in cardiomyocytes via NF-kappa B activation. Chandrasekar, B., Colston, J.T., de la Rosa, S.D., Rao, P.P., Freeman, G.L. Biochem. Biophys. Res. Commun. (2003) [Pubmed]
  11. Fractalkine (CX3CL1) stimulated by nuclear factor kappaB (NF-kappaB)-dependent inflammatory signals induces aortic smooth muscle cell proliferation through an autocrine pathway. Chandrasekar, B., Mummidi, S., Perla, R.P., Bysani, S., Dulin, N.O., Liu, F., Melby, P.C. Biochem. J. (2003) [Pubmed]
  12. Calorie restriction attenuates inflammatory responses to myocardial ischemia-reperfusion injury. Chandrasekar, B., Nelson, J.F., Colston, J.T., Freeman, G.L. Am. J. Physiol. Heart Circ. Physiol. (2001) [Pubmed]
  13. Ischemia-reperfusion of rat myocardium activates nuclear factor-KappaB and induces neutrophil infiltration via lipopolysaccharide-induced CXC chemokine. Chandrasekar, B., Smith, J.B., Freeman, G.L. Circulation (2001) [Pubmed]
 
WikiGenes - Universities