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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Edward J. Calabrese

Department of Public Health

Environmental Health Sciences

University of Massachusetts

Amherst

USA

[email]@*.umass.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Public Health, Environmental Health Sciences, University of Massachusetts, Amherst, USA. 1998 - 2010
  • UMASS School of Public Health and Health Sciences, Amherst, USA. 2003 - 2004

References

  1. Dose response biology: the case of resveratrol. Calabrese, E.J., Mattson, M.P., Calabrese, V. Hum. Exp. Toxicol (2010) [Pubmed]
  2. Resveratrol commonly displays hormesis: occurrence and biomedical significance. Calabrese, E.J., Mattson, M.P., Calabrese, V. Hum. Exp. Toxicol (2010) [Pubmed]
  3. Resveratrol: an assessment of its dose response an introduction. Calabrese, E.J. Hum. Exp. Toxicol (2010) [Pubmed]
  4. Tumor resistance explained by hormesis. Calabrese, E.J., Nascarella, M.A. Dose. Response (2010) [Pubmed]
  5. Hormesis in high-throughput screening of antibacterial compounds in E coli. Calabrese, E.J., Hoffmann, G.R., Stanek, E.J., Nascarella, M.A. Hum. Exp. Toxicol (2010) [Pubmed]
  6. Toxicity testing in the 21st century--a view from BELLE. Calabrese, E.J. Hum. Exp. Toxicol (2010) [Pubmed]
  7. BELLE: an evolving legacy. Calabrese, E.J. Hum. Exp. Toxicol (2010) [Pubmed]
  8. Hormesis is central to toxicology, pharmacology and risk assessment. Calabrese, E.J. Hum. Exp. Toxicol (2010) [Pubmed]
  9. Hormesis: calabrese responds. Calabrese, E.J. Environ. Health Perspect. (2010) [Pubmed]
  10. Hormesis and plant biology. Calabrese, E.J., Blain, R.B. Environ. Pollut. (2009) [Pubmed]
  11. Getting the dose-response wrong: why hormesis became marginalized and the threshold model accepted. Calabrese, E.J. Arch. Toxicol. (2009) [Pubmed]
  12. The road to linearity: why linearity at low doses became the basis for carcinogen risk assessment. Calabrese, E.J. Arch. Toxicol. (2009) [Pubmed]
  13. Hormesis, non-linearity, and risk communication. Calabrese, E. Hum. Exp. Toxicol (2009) [Pubmed]
  14. Hormesis: a conversation with a critic. Calabrese, E.J. Environ. Health Perspect. (2009) [Pubmed]
  15. Astrocytes: adaptive responses to low doses of neurotoxins. Calabrese, E.J. Crit. Rev. Toxicol. (2008) [Pubmed]
  16. Alzheimer's disease drugs: an application of the hormetic dose-response model. Calabrese, E.J. Crit. Rev. Toxicol. (2008) [Pubmed]
  17. Hormesis predicts low-dose responses better than threshold models. Calabrese, E.J., Stanek, E.J., Nascarella, M.A., Hoffmann, G.R. Int. J. Toxicol. (2008) [Pubmed]
  18. Converging concepts: adaptive response, preconditioning, and the Yerkes-Dodson Law are manifestations of hormesis. Calabrese, E.J. Ageing Res. Rev. (2008) [Pubmed]
  19. Hormesis: why it is important to toxicology and toxicologists. Calabrese, E.J. Environ. Toxicol. Chem. (2008) [Pubmed]
  20. Another California milestone: the first application of hormesis in litigation and regulation. Calabrese, E.J. Int. J. Toxicol. (2008) [Pubmed]
  21. Neuroscience and hormesis: overview and general findings. Calabrese, E.J. Crit. Rev. Toxicol. (2008) [Pubmed]
  22. Dose-response features of neuroprotective agents: an integrative summary. Calabrese, E.J. Crit. Rev. Toxicol. (2008) [Pubmed]
  23. Pharmacological enhancement of neuronal survival. Calabrese, E.J. Crit. Rev. Toxicol. (2008) [Pubmed]
  24. Enhancing and regulating neurite outgrowth. Calabrese, E.J. Crit. Rev. Toxicol. (2008) [Pubmed]
  25. Hormesis and the law: introduction. Calabrese, E.J. Hum. Exp. Toxicol (2008) [Pubmed]
  26. Stress biology and hormesis: the Yerkes-Dodson law in psychology--a special case of the hormesis dose response. Calabrese, E.J. Crit. Rev. Toxicol. (2008) [Pubmed]
  27. P-glycoprotein efflux transporter activity often displays biphasic dose-response relationships. Calabrese, E.J. Crit. Rev. Toxicol. (2008) [Pubmed]
  28. An assessment of anxiolytic drug screening tests: hormetic dose responses predominate. Calabrese, E.J. Crit. Rev. Toxicol. (2008) [Pubmed]
  29. Modulation of the epileptic seizure threshold: implications of biphasic dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2008) [Pubmed]
  30. Hormesis and medicine. Calabrese, E.J. Br. J. Clin. Pharmacol (2008) [Pubmed]
  31. Pain and u-shaped dose responses: occurrence, mechanisms, and clinical implications. Calabrese, E.J. Crit. Rev. Toxicol. (2008) [Pubmed]
  32. U-shaped dose response in behavioral pharmacology: historical foundations. Calabrese, E.J. Crit. Rev. Toxicol. (2008) [Pubmed]
  33. Addiction and dose response: the psychomotor stimulant theory of addiction reveals that hormetic dose responses are dominant. Calabrese, E.J. Crit. Rev. Toxicol. (2008) [Pubmed]
  34. Hormesis and ethics: introduction. Calabrese, E. Hum. Exp. Toxicol (2008) [Pubmed]
  35. Biological stress response terminology: Integrating the concepts of adaptive response and preconditioning stress within a hormetic dose-response framework. Calabrese, E.J., Bachmann, K.A., Bailer, A.J., Bolger, P.M., Borak, J., Cai, L., Cedergreen, N., Cherian, M.G., Chiueh, C.C., Clarkson, T.W., Cook, R.R., Diamond, D.M., Doolittle, D.J., Dorato, M.A., Duke, S.O., Feinendegen, L., Gardner, D.E., Hart, R.W., Hastings, K.L., Hayes, A.W., Hoffmann, G.R., Ives, J.A., Jaworowski, Z., Johnson, T.E., Jonas, W.B., Kaminski, N.E., Keller, J.G., Klaunig, J.E., Knudsen, T.B., Kozumbo, W.J., Lettieri, T., Liu, S.Z., Maisseu, A., Maynard, K.I., Masoro, E.J., McClellan, R.O., Mehendale, H.M., Mothersill, C., Newlin, D.B., Nigg, H.N., Oehme, F.W., Phalen, R.F., Philbert, M.A., Rattan, S.I., Riviere, J.E., Rodricks, J., Sapolsky, R.M., Scott, B.R., Seymour, C., Sinclair, D.A., Smith-Sonneborn, J., Snow, E.T., Spear, L., Stevenson, D.E., Thomas, Y., Tubiana, M., Williams, G.M., Mattson, M.P. Toxicol. Appl. Pharmacol. (2007) [Pubmed]
  36. Threshold Dose--Response Model--RIP: 1911 to 2006. Calabrese, E.J. Bioessays (2007) [Pubmed]
  37. Elliott's ethics of expertise proposal and application: a dangerous precedent. Calabrese, E.J. Sci. Eng. Ethics (2007) [Pubmed]
  38. Drug development and hormesis: changing conceptual understanding of the dose response creates new challenges and opportunities for more effective drugs. Calabrese, E.J., Staudenmayer, J.W., Stanek, E.J. Curr. Opin. Drug. Discov. Devel (2006) [Pubmed]
  39. The failure of dose-response models to predict low dose effects: a major challenge for biomedical, toxicological and aging research. Calabrese, E.J. Biogerontology (2006) [Pubmed]
  40. Hormesis outperforms threshold model in National Cancer Institute antitumor drug screening database. Calabrese, E.J., Staudenmayer, J.W., Stanek, E.J., Hoffmann, G.R. Toxicol. Sci. (2006) [Pubmed]
  41. The occurrence of hormetic dose responses in the toxicological literature, the hormesis database: an overview. Calabrese, E.J., Blain, R. Toxicol. Appl. Pharmacol. (2005) [Pubmed]
  42. Toxicological awakenings: the rebirth of hormesis as a central pillar of toxicology. Calabrese, E.J. Toxicol. Appl. Pharmacol. (2005) [Pubmed]
  43. Hormetic dose-response relationships in immunology: occurrence, quantitative features of the dose response, mechanistic foundations, and clinical implications. Calabrese, E.J. Crit. Rev. Toxicol. (2005) [Pubmed]
  44. Hormesis: how it could affect the risk assessment process. Calabrese, E.J., Cook, R.R. Hum. Exp. Toxicol (2005) [Pubmed]
  45. Paradigm lost, paradigm found: the re-emergence of hormesis as a fundamental dose response model in the toxicological sciences. Calabrese, E.J. Environ. Pollut. (2005) [Pubmed]
  46. Historical blunders: how toxicology got the dose-response relationship half right. Calabrese, E.J. Cell. Mol. Biol. (Noisy-le-grand) (2005) [Pubmed]
  47. Cancer biology and hormesis: human tumor cell lines commonly display hormetic (biphasic) dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2005) [Pubmed]
  48. Hormesis: from marginalization to mainstream: a case for hormesis as the default dose-response model in risk assessment. Calabrese, E.J. Toxicol. Appl. Pharmacol. (2004) [Pubmed]
  49. Hormesis: a revolution in toxicology, risk assessment and medicine. Calabrese, E.J. EMBO Rep. (2004) [Pubmed]
  50. Hormesis: the dose-response revolution. Calabrese, E.J., Baldwin, L.A. Annu. Rev. Pharmacol. Toxicol. (2003) [Pubmed]
  51. The hormetic dose-response model is more common than the threshold model in toxicology. Calabrese, E.J., Baldwin, L.A. Toxicol. Sci. (2003) [Pubmed]
  52. Risk communication and the challenge of hormesis. Calabrese, E. Hum. Exp. Toxicol (2003) [Pubmed]
  53. The effects of diisopropylmethylphosphonate, a by-product of the production of sarin and a contaminant in drinking water at the Rocky Mountain Arsenal, on female mink. Calabrese, E.J. Regul. Toxicol. Pharmacol. (2003) [Pubmed]
  54. Inorganics and hormesis. Calabrese, E.J., Baldwin, L.A. Crit. Rev. Toxicol. (2003) [Pubmed]
  55. Chemotherapeutics and hormesis. Calabrese, E.J., Baldwin, L.A. Crit. Rev. Toxicol. (2003) [Pubmed]
  56. Peptides and hormesis. Calabrese, E.J., Baldwin, L.A. Crit. Rev. Toxicol. (2003) [Pubmed]
  57. Ethanol and hormesis. Calabrese, E.J., Baldwin, L.A. Crit. Rev. Toxicol. (2003) [Pubmed]
  58. The effects of diisopropylmethylphosphonate on female mink: how medical intervention biased mortality data. Calabrese, E.J. Regul. Toxicol. Pharmacol. (2003) [Pubmed]
  59. Hormesis: changing view of the dose-response, a personal account of the history and current status. Calabrese, E.J. Mutat. Res. (2002) [Pubmed]
  60. Defining hormesis. Calabrese, E.J., Baldwin, L.A. Hum. Exp. Toxicol (2002) [Pubmed]
  61. Applications of hormesis in toxicology, risk assessment and chemotherapeutics. Calabrese, E.J., Baldwin, L.A. Trends Pharmacol. Sci. (2002) [Pubmed]
  62. Hormesis and high-risk groups. Calabrese, E.J., Baldwin, L.A. Regul. Toxicol. Pharmacol. (2002) [Pubmed]
  63. U-shaped dose-responses in biology, toxicology, and public health. Calabrese, E.J., Baldwin, L.A. Annu. Rev. Public. Health (2001) [Pubmed]
  64. Hormesis: U-shaped dose responses and their centrality in toxicology. Calabrese, E.J., Baldwin, L.A. Trends Pharmacol. Sci. (2001) [Pubmed]
  65. The frequency of U-shaped dose responses in the toxicological literature. Calabrese, E.J., Baldwin, L.A. Toxicol. Sci. (2001) [Pubmed]
  66. Hormesis: a generalizable and unifying hypothesis. Calabrese, E.J., Baldwin, L.A. Crit. Rev. Toxicol. (2001) [Pubmed]
  67. Overcompensation stimulation: a mechanism for hormetic effects. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  68. Agonist concentration gradients as a generalizable regulatory implementation strategy. Calabrese, E.J., Baldwin, L.A. Crit. Rev. Toxicol. (2001) [Pubmed]
  69. Prostaglandins: biphasic dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  70. Nitric oxide: biphasic dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  71. Estrogen and related compounds: biphasic dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  72. Androgens: biphasic dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  73. Adrenergic receptors: biphasic dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  74. Adenosine: biphasic dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  75. 5-Hydroxytryptamine (serotonin): biphasic dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  76. Dopamine: biphasic dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  77. Opiates: biphasic dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  78. Amyloid beta-peptide: biphasic dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  79. Apoptosis: biphasic dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  80. Cell migration/chemotaxis: biphasic dose responses. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  81. The future of hormesis: where do we go from here?. Calabrese, E.J. Crit. Rev. Toxicol. (2001) [Pubmed]
  82. The effects of gamma rays on longevity. Calabrese, E.J., Baldwin, L.A. Biogerontology (2000) [Pubmed]
  83. The Single Exposure Carcinogen Database: assessing the circumstances under which a single exposure to a carcinogen can cause cancer. Calabrese, E.J., Blain, R.B. Toxicol. Sci. (1999) [Pubmed]
  84. Hormesis as a biological hypothesis. Calabrese, E.J., Baldwin, L.A. Environ. Health Perspect. (1998) [Pubmed]
 
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