H.C. Kirch
Department of Internal Medicine (Cancer Research)
University of Essen
Germany
Name/email consistency: high
- Tumor-specific activation of hTERT-derived promoters by tumor suppressive E1A-mutants involves recruitment of p300/CBP/HAT and suppression of HDAC-1 and defines a combined tumor targeting and suppression system. Kirch, H.C., Ruschen, S., Brockmann, D., Esche, H., Horikawa, I., Barrett, J.C., Opalka, B., Hengge, U.R. Oncogene (2002)
- Formation of the early-region-2 transcription-factor-1-retinoblastoma-protein (E2F-1-RB) transrepressor and release of the retinoblastoma protein from nuclear complexes containing cyclin A is induced by interferon alpha in U937V cells but not in interferon-alpha-resistant U937VR cells. Kirch, H.C., Putzer, B., Brockmann, D., Esche, H., Kloke, O. Eur. J. Biochem. (1997)