Grant M. Hatch
Dept. of Pharmacology and Therapeutics
753 McDermot Ave.
Faculty of Medicine
Univ. of Manitoba
Name/email consistency: high
- Persistent pulmonary hypertension results in reduced tetralinoleoyl-cardiolipin and mitochondrial complex II + III during the development of right ventricular hypertrophy in the neonatal pig heart. Saini-Chohan, H.K., Dakshinamurti, S., Taylor, W.A., Shen, G.X., Murphy, R., Sparagna, G.C., Hatch, G.M. Am. J. Physiol. Heart Circ. Physiol. (2011)
- C. trachomatis-infection accelerates metabolism of phosphatidylcholine derived from low density lipoprotein but does not affect phosphatidylcholine secretion from hepatocytes. Hatch, G.M., McClarty, G. BMC Microbiol. (2004)
- Cell biology of cardiac mitochondrial phospholipids. Hatch, G.M. Biochem. Cell Biol. (2004)
- AGI-1067. AtheroGenics. Hatch, G.M. Curr. Opin. Investig. Drugs (2002)
- Cardiolipin remodeling in eukaryotic cells infected with Chlamydia trachomatis is linked to elevated mitochondrial metabolism. Hatch, G.M., McClarty, G. Biochem. Biophys. Res. Commun. (1998)
The world's first wiki where authorship really matters (Nature Genetics, 2008). Due credit and reputation for authors. Imagine a global collaborative knowledge base for original thoughts. Search thousands of articles and collaborate with scientists around the globe.








