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Hermann Lage

Institute of Pathology

Charité Campus Mitte

[email]@charite.de

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Institute of Pathology, Charité Campus Mitte. 2010
  • Charité Campus Mitte, Institute of Pathology, Berlin, Germany. 2003 - 2009
  • Humboldt-University Berlin, University Hospital Charité Campus Mitte, Institute of Pathology, Germany. 1999 - 2004
  • Institute of Pathology, Charité Campus Mitte, Humboldt University Berlin, Germany. 1999 - 2001

References

  1. Bacterial delivery of RNAi effectors: transkingdom RNAi. Lage, H., Krühn, A. J. Vis. Exp (2010) [Pubmed]
  2. Therapeutic potential of RNA interference in drug-resistant cancers. Lage, H. Future. Oncol (2009) [Pubmed]
  3. An overview of cancer multidrug resistance: a still unsolved problem. Lage, H. Cell. Mol. Life Sci. (2008) [Pubmed]
  4. High antineoplastic activity of new heterocyclic compounds in cancer cells with resistance against classical DNA topoisomerase II-targeting drugs. Lage, H., Aki-Sener, E., Yalcin, I. Int. J. Cancer (2006) [Pubmed]
  5. MDR1/P-glycoprotein (ABCB1) as target for RNA interference-mediated reversal of multidrug resistance. Lage, H. Curr. Drug. Targets (2006) [Pubmed]
  6. Potential applications of RNA interference technology in the treatment of cancer. Lage, H. Future. Oncology. (london,. England) (2005) [Pubmed]
  7. Proteomics in cancer cell research: an analysis of therapy resistance. Lage, H. Pathol. Res. Pract. (2004) [Pubmed]
  8. ABC-transporters: implications on drug resistance from microorganisms to human cancers. Lage, H. Int. J. Antimicrob. Agents (2003) [Pubmed]
  9. Molecular analysis of therapy resistance in gastric cancer. Lage, H. Dig. Dis (2003) [Pubmed]
  10. Multiple mechanisms confer different drug-resistant phenotypes in pancreatic carcinoma cells. Lage, H., Dietel, M. J. Cancer Res. Clin. Oncol. (2002) [Pubmed]
  11. DFNA5 (ICERE-1) contributes to acquired etoposide resistance in melanoma cells. Lage, H., Helmbach, H., Grottke, C., Dietel, M., Schadendorf, D. FEBS Lett. (2001) [Pubmed]
  12. Expression of a glypican-related 62-kDa antigen is decreased in hepatocellular carcinoma in correspondence to the grade of tumor differentiation. Lage, H., Kellner, U., Tannapfel, A., Dietel, M. Virchows Arch. (2001) [Pubmed]
  13. Enhanced expression of human ABC-transporter tap is associated with cellular resistance to mitoxantrone. Lage, H., Perlitz, C., Abele, R., Tampé, R., Dietel, M., Schadendorf, D., Sinha, P. FEBS Lett. (2001) [Pubmed]
  14. Thermosensitivity of multidrug-resistant human gastric and pancreatic carcinoma cells. Lage, H., Jordan, A., Scholz, R., Dietel, M. Int. J. Hyperthermia (2000) [Pubmed]
  15. Effect of the breast-cancer resistance protein on atypical multidrug resistance. Lage, H., Dietel, M. Lancet Oncol. (2000) [Pubmed]
  16. Expression of DNA repair proteins hMSH2, hMSH6, hMLH1, O6-methylguanine-DNA methyltransferase and N-methylpurine-DNA glycosylase in melanoma cells with acquired drug resistance. Lage, H., Christmann, M., Kern, M.A., Dietel, M., Pick, M., Kaina, B., Schadendorf, D. Int. J. Cancer (1999) [Pubmed]
  17. Involvement of the DNA mismatch repair system in antineoplastic drug resistance. Lage, H., Dietel, M. J. Cancer Res. Clin. Oncol. (1999) [Pubmed]
 
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