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MeSH Review

Spinal Osteophytosis

 
 
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Disease relevance of Spinal Osteophytosis

 

Psychiatry related information on Spinal Osteophytosis

 

High impact information on Spinal Osteophytosis

  • Other contributory factors include coexisting spondylosis, a congenitally narrow canal, friction between cord and OPLL during neck movement and acute trauma [7].
  • There was no correlation between delta BMD and any of the lifestyle, muscle strength, dietary, or hormonal indices or with the severity of spinal osteophytosis [8].
  • The mean concentration of immunoreactive CRH in the cerebrospinal fluid of seven patients was 30.8 +/- 3.9 pg/ml (+/- SEM), which was higher than the value of 18.4 +/- 1.1 pg/ml (P less than 0.005) in control subjects with cervical spondylosis [9].
  • These findings indicate that the klotho gene may be a candidate for the genetic regulation of common age-related diseases like osteoporosis and spondylosis, and we provide the first evidence suggesting that this gene may be involved in the etiology of human diseases [10].
  • Most of these patients had cervical spondylosis or rheumatoid arthritis involving the cervical spine [11].
 

Chemical compound and disease context of Spinal Osteophytosis

 

Biological context of Spinal Osteophytosis

 

Anatomical context of Spinal Osteophytosis

 

Gene context of Spinal Osteophytosis

 

Analytical, diagnostic and therapeutic context of Spinal Osteophytosis

References

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