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MeSH Review

Kidney Calculi

 
 
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Disease relevance of Kidney Calculi

 

Psychiatry related information on Kidney Calculi

 

High impact information on Kidney Calculi

 

Chemical compound and disease context of Kidney Calculi

 

Biological context of Kidney Calculi

 

Anatomical context of Kidney Calculi

 

Gene context of Kidney Calculi

  • Concentrations and relative amounts of TFF1 in the urine of patients with idiopathic CaOx kidney stone were significantly less (2.5-fold for the concentrations and 5- to 22-fold for the relative amounts) than those found in controls [25].
  • Characterisation of renal chloride channel, CLCN5, mutations in hypercalciuric nephrolithiasis (kidney stones) disorders [26].
  • We find trpm7 expression in the mesonephric kidney and show that mutants develop kidney stones, indicating renal dysfunction [27].
  • 5. The protein encoded by ORG has a similar pattern of expression and identical structural features to nephrocalcin, a calcium-binding protein partially purified from kidney that plays a role in calcium reabsorption and in prevention of nephrolithiasis [28].
  • Mutations in the CLCN5 gene have been demonstrated in three disorders of hypercalciuric nephrolithiasis, i.e., Dent's disease, X-linked recessive nephrolithiasis, and X-linked recessive hypophosphatemic rickets [29].
 

Analytical, diagnostic and therapeutic context of Kidney Calculi

References

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  22. An acidic peptide sequence of nucleolin-related protein can mediate the attachment of calcium oxalate to renal tubule cells. Sorokina, E.A., Wesson, J.A., Kleinman, J.G. J. Am. Soc. Nephrol. (2004) [Pubmed]
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  28. The mouse osteocalcin gene cluster contains three genes with two separate spatial and temporal patterns of expression. Desbois, C., Hogue, D.A., Karsenty, G. J. Biol. Chem. (1994) [Pubmed]
  29. Mutations in CLCN5 chloride channel in Japanese patients with low molecular weight proteinuria. Morimoto, T., Uchida, S., Sakamoto, H., Kondo, Y., Hanamizu, H., Fukui, M., Tomino, Y., Nagano, N., Sasaki, S., Marumo, F. J. Am. Soc. Nephrol. (1998) [Pubmed]
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  32. Direct correlation between hyperoxaluria/oxalate stone disease and the absence of the gastrointestinal tract-dwelling bacterium Oxalobacter formigenes: possible prevention by gut recolonization or enzyme replacement therapy. Sidhu, H., Schmidt, M.E., Cornelius, J.G., Thamilselvan, S., Khan, S.R., Hesse, A., Peck, A.B. J. Am. Soc. Nephrol. (1999) [Pubmed]
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