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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
 
 
 
 

Lesion-induced sprouting in the rat dentate gyrus is inhibited by repeated ethanol administration.

The effect of ethanol on hippocampal axonal sprouting was studied with a histochemical technique for identifying acetylcholinesterase. Unilateral lesion of the entorhinal cortex in adult rats produced an increase in the density of acetylcholinesterase staining in the outer molecular layer and a concomitant increase in the width of the pale-staining commissural-associational zone of the dentate gyrus. Other rats were given ethanol (11.3 +/- 0.45 grams per kilogram) for 2 weeks before and 9 days after receiving the lesion. Ethanol abolished the expansion of the commissural-associated zone. The effect of ethanol on sprouting axons suggests that it may inhibit recovery of function after brain injury.[1]

References

  1. Lesion-induced sprouting in the rat dentate gyrus is inhibited by repeated ethanol administration. West, J.R., Lind, M.D., Demuth, R.M., Parker, E.S., Alkana, R.L., Cassell, M., Black, A.C. Science (1982) [Pubmed]
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