Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

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Institute of Molecular Biology and Biochemistry

UKBF, FU Berlin

Berlin

Germany

Name/email consistency: high

Institute of Molecular Biology and Biochemistry, UKBF, FU Berlin, Berlin, Germany. 1997 - 2002
Institut für Molekularbiologie und Biochemie und Centrum Somatische Gentherapie, FU, Berlin, Germany. 1999
Department of Dermatology, Virchow-Clinic, Humboldt University, Berlin, Germany. 1996

Proliferation-dependent induction of apoptosis by the retinoid CD437 in p53-mutated keratinocytes. Wanner, R., Henseleit-Walter, U., Wittig, B., Kolde, G. J. Mol. Med. (2002) [Pubmed]
The loss of desmosomes after retinoic acid treatment results in an apparent inhibition of HaCaT keratinocyte differentiation. Wanner, R., Wolff, B., Glowacki, F., Kolde, G., Wittig, B. Arch. Dermatol. Res. (1999) [Pubmed]
Polymorphism at codon 554 of the human Ah receptor: different allelic frequencies in Caucasians and Japanese and no correlation with severity of TCDD induced chloracne in chemical workers. Wanner, R., Zober, A., Abraham, K., Kleffe, J., Henz, B.M., Wittig, B. Pharmacogenetics (1999) [Pubmed]
The promoter of the HaCaT keratinocyte differentiation-related gene keratin 4 contains a functional AP-2 binding site. Wanner, R., Zhang, J., Dorbic, T., Mischke, D., Henz, B.M., Wittig, B., Rosenbach, T. Arch. Dermatol. Res. (1997) [Pubmed]
Retinoic acid affects the expression rate of the differentiation-related genes aryl hydrocarbon receptor, ARNT and keratin 4 in proliferative keratinocytes only. Wanner, R., Panteleyev, A., Henz, B.M., Rosenbach, T. Biochim. Biophys. Acta (1996) [Pubmed]