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N.M. Sawtell

Division of Infectious Diseases

Children's Hospital Medical Center

Cincinnati

Ohio 45229-3039

USA

[email]@chmcc.org

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Affiliation

Division of Infectious Diseases, Children's Hospital Medical Center, Cincinnati, Ohio 45229-3039, USA. 1998 - 2006

References

Herpes simplex virus DNA synthesis is not a decisive regulatory event in the initiation of lytic viral protein expression in neurons in vivo during primary infection or reactivation from latency. Sawtell, N.M., Thompson, R.L., Haas, R.L. J. Virol. (2006) [Pubmed]
Comparison of herpes simplex virus reactivation in ganglia in vivo and in explants demonstrates quantitative and qualitative differences. Sawtell, N.M., Thompson, R.L. J. Virol. (2004) [Pubmed]
Quantitative analysis of herpes simplex virus reactivation in vivo demonstrates that reactivation in the nervous system is not inhibited at early times postinoculation. Sawtell, N.M. J. Virol. (2003) [Pubmed]
Early intervention with high-dose acyclovir treatment during primary herpes simplex virus infection reduces latency and subsequent reactivation in the nervous system in vivo. Sawtell, N.M., Thompson, R.L., Stanberry, L.R., Bernstein, D.I. J. Infect. Dis. (2001) [Pubmed]
A temporal analysis of acyclovir inhibition of induced herpes simplex virus type 1 In vivo reactivation in the mouse trigeminal ganglia. Sawtell, N.M., Bernstein, D.I., Stanberry, L.R. J. Infect. Dis. (1999) [Pubmed]
The probability of in vivo reactivation of herpes simplex virus type 1 increases with the number of latently infected neurons in the ganglia. Sawtell, N.M. J. Virol. (1998) [Pubmed]