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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Sergei N. Rodin

Human Genetics Center

School of Public Health

University of Texas

Houston

USA

[email]@coh.org

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Human Genetics Center, School of Public Health, University of Texas, Houston, USA. 2009
  • Theoretical Biology Department, Beckman Research Institute of the City of Hope, Duarte, CA 91010-3000, USA. 2002 - 2008

References

  1. On primordial sense-antisense coding. Rodin, A.S., Rodin, S.N., Carter, C.W. J. Mol. Evol. (2009) [Pubmed]
  2. On the origin of the genetic code: signatures of its primordial complementarity in tRNAs and aminoacyl-tRNA synthetases. Rodin, S.N., Rodin, A.S. Heredity (2008) [Pubmed]
  3. Origin of the genetic code: first aminoacyl-tRNA synthetases could replace isofunctional ribozymes when only the second base of codons was established. Rodin, S.N., Rodin, A.S. DNA Cell Biol. (2006) [Pubmed]
  4. Partitioning of aminoacyl-tRNA synthetases in two classes could have been encoded in a strand-symmetric RNA world. Rodin, S.N., Rodin, A.S. DNA Cell Biol. (2006) [Pubmed]
  5. Origins and selection of p53 mutations in lung carcinogenesis. Rodin, S.N., Rodin, A.S. Semin. Cancer Biol. (2005) [Pubmed]
  6. Repositioning-dependent fate of duplicate genes. Rodin, S.N., Parkhomchuk, D.V., Rodin, A.S., Holmquist, G.P., Riggs, A.D. DNA Cell Biol. (2005) [Pubmed]
  7. Position-associated GC asymmetry of gene duplicates. Rodin, S.N., Parkhomchuk, D.V. J. Mol. Evol. (2004) [Pubmed]
  8. Epigenetic silencing may aid evolution by gene duplication. Rodin, S.N., Riggs, A.D. J. Mol. Evol. (2003) [Pubmed]
  9. On the origin of p53 G:C --> T:A transversions in lung cancers. Rodin, S.N., Rodin, A.S. Mutat. Res. (2002) [Pubmed]
  10. Cancerous hyper-mutagenesis in p53 genes is possibly associated with transcriptional bypass of DNA lesions. Rodin, S.N., Rodin, A.S., Juhasz, A., Holmquist, G.P. Mutat. Res. (2002) [Pubmed]
 
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