Toshiharu Oba
Department of Cell Physiology
Nagoya City University Graduate School of Medical Sciences
Mizuho-ku
Nagoya
Japan
Name/email consistency: high
- Cellular redox state protects acetaldehyde-induced alteration in cardiomyocyte function by modifying Ca2+ release from sarcoplasmic reticulum. Oba, T., Maeno, Y., Nagao, M., Sakuma, N., Murayama, T. Am. J. Physiol. Heart Circ. Physiol. (2008)
- Differential contribution of clinical amounts of acetaldehyde to skeletal and cardiac muscle dysfunction in alcoholic myopathy. Oba, T., Maeno, Y., Ishida, K. Curr. Pharm. Des. (2005)
- Acetaldehyde alters Ca2+-release channel gating and muscle contraction in a dose-dependent manner. Oba, T., Maeno, Y. Am. J. Physiol., Cell Physiol. (2004)
- Redox states of type 1 ryanodine receptor alter Ca(2+) release channel response to modulators. Oba, T., Murayama, T., Ogawa, Y. Am. J. Physiol., Cell Physiol. (2002)
- H2O2 activates ryanodine receptor but has little effect on recovery of releasable Ca2+ content after fatigue. Oba, T., Kurono, C., Nakajima, R., Takaishi, T., Ishida, K., Fuller, G.A., Klomkleaw, W., Yamaguchi, M. J. Appl. Physiol. (2002)
- Hydrogen peroxide decelerates recovery of action potential after high-frequency fatigue in skeletal muscle. Oba, T., Ishikawa, T., Takaishi, T., Aoki, T., Yamaguchi, M. Muscle. Nerve (2000)
- H(2)O(2) and ethanol act synergistically to gate ryanodine receptor/calcium-release channel. Oba, T., Ishikawa, T., Murayama, T., Ogawa, Y., Yamaguchi, M. Am. J. Physiol., Cell Physiol. (2000)
- Different effects of two gold compounds on muscle contraction, membrane potential and ryanodine receptor. Oba, T., Ishikawa, T., Yamaguchi, M. Eur. J. Pharmacol. (1999)
