Tony Wyss-Coray
Gladstone Institute of Neurological Disease
San Francisco
California 94141-9100
USA
Name/email consistency: high
- Prominent neurodegeneration and increased plaque formation in complement-inhibited Alzheimer's mice. Wyss-Coray, T., Yan, F., Lin, A.H., Lambris, J.D., Alexander, J.J., Quigg, R.J., Masliah, E. Proc. Natl. Acad. Sci. U.S.A. (2002)
- Inflammation in neurodegenerative disease--a double-edged sword. Wyss-Coray, T., Mucke, L. Neuron (2002)
- TGF-beta1 promotes microglial amyloid-beta clearance and reduces plaque burden in transgenic mice. Wyss-Coray, T., Lin, C., Yan, F., Yu, G.Q., Rohde, M., McConlogue, L., Masliah, E., Mucke, L. Nat. Med. (2001)
- Key signaling pathways regulate the biological activities and accumulation of amyloid-beta. Wyss-Coray, T., McConlogue, L., Kindy, M., Schmidt, A.M., Du Yan, S., Stern, D.M. Neurobiol. Aging (2001)
- Chronic overproduction of transforming growth factor-beta1 by astrocytes promotes Alzheimer's disease-like microvascular degeneration in transgenic mice. Wyss-Coray, T., Lin, C., Sanan, D.A., Mucke, L., Masliah, E. Am. J. Pathol. (2000)
- Alzheimer's disease-like cerebrovascular pathology in transforming growth factor-beta 1 transgenic mice and functional metabolic correlates. Wyss-Coray, T., Lin, C., von Euw, D., Masliah, E., Mucke, L., Lacombe, P. Ann. N. Y. Acad. Sci. (2000)