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Tony Wyss-Coray

Gladstone Institute of Neurological Disease

San Francisco

California 94141-9100

USA

[email]@*.ucsf.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Gladstone Institute of Neurological Disease, San Francisco, California 94141-9100, USA. 2000 - 2002
  • Gladstone Institute of Neurological Disease and Department of Neurology, University of California, San Francisco, CA 94141, USA. 2001 - 2002

References

  1. Prominent neurodegeneration and increased plaque formation in complement-inhibited Alzheimer's mice. Wyss-Coray, T., Yan, F., Lin, A.H., Lambris, J.D., Alexander, J.J., Quigg, R.J., Masliah, E. Proc. Natl. Acad. Sci. U.S.A. (2002) [Pubmed]
  2. Inflammation in neurodegenerative disease--a double-edged sword. Wyss-Coray, T., Mucke, L. Neuron (2002) [Pubmed]
  3. TGF-beta1 promotes microglial amyloid-beta clearance and reduces plaque burden in transgenic mice. Wyss-Coray, T., Lin, C., Yan, F., Yu, G.Q., Rohde, M., McConlogue, L., Masliah, E., Mucke, L. Nat. Med. (2001) [Pubmed]
  4. Key signaling pathways regulate the biological activities and accumulation of amyloid-beta. Wyss-Coray, T., McConlogue, L., Kindy, M., Schmidt, A.M., Du Yan, S., Stern, D.M. Neurobiol. Aging (2001) [Pubmed]
  5. Chronic overproduction of transforming growth factor-beta1 by astrocytes promotes Alzheimer's disease-like microvascular degeneration in transgenic mice. Wyss-Coray, T., Lin, C., Sanan, D.A., Mucke, L., Masliah, E. Am. J. Pathol. (2000) [Pubmed]
  6. Alzheimer's disease-like cerebrovascular pathology in transforming growth factor-beta 1 transgenic mice and functional metabolic correlates. Wyss-Coray, T., Lin, C., von Euw, D., Masliah, E., Mucke, L., Lacombe, P. Ann. N. Y. Acad. Sci. (2000) [Pubmed]
 
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