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Yohannes Tesfaigzi

Lovelace Respiratory Research Institute

2425 Ridgecrest Dr. SE

Albuquerque

NM 87108

[email]@lrri.org

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Lovelace Respiratory Research Institute, 2425 Ridgecrest Dr. SE, Albuquerque, NM 87108. 2000 - 2012

References

  1. Acute Inflammation Induces Insulin-like Growth Factor-1 to Mediate Bcl-2 and Muc5ac Expression in Airway Epithelial Cells. Chand, H.S., Woldegiorgis, Z., Schwalm, K., McDonald, J., Tesfaigzi, Y. Am. J. Respir. Cell Mol. Biol. (2012) [Pubmed]
  2. New mexican Hispanic smokers have lower odds of chronic obstructive pulmonary disease and less decline in lung function than non-Hispanic whites. Bruse, S., Sood, A., Petersen, H., Liu, Y., Leng, S., Celedón, J.C., Gilliland, F., Celli, B., Belinsky, S.A., Tesfaigzi, Y. Am. J. Respir. Crit. Care Med. (2011) [Pubmed]
  3. Cigarette smoke suppresses bik to cause epithelial cell hyperplasia and mucous cell metaplasia. Mebratu, Y.A., Schwalm, K., Smith, K.R., Schuyler, M., Tesfaigzi, Y. Am. J. Respir. Crit. Care Med. (2011) [Pubmed]
  4. Regulation of mucous cell metaplasia in bronchial asthma. Tesfaigzi, Y. Curr. Mol. Med. (2008) [Pubmed]
  5. Roles of apoptosis in airway epithelia. Tesfaigzi, Y. Am. J. Respir. Cell Mol. Biol. (2006) [Pubmed]
  6. Genotypes in matrix metalloproteinase 9 are a risk factor for COPD. Tesfaigzi, Y., Myers, O.B., Stidley, C.A., Schwalm, K., Picchi, M., Crowell, R.E., Gilliland, F.D., Belinsky, S.A. International J. Chronic Obstructive Pulmonary Disease (2006) [Pubmed]
  7. Low-level subchronic exposure to wood smoke exacerbates inflammatory responses in allergic rats. Tesfaigzi, Y., McDonald, J.D., Reed, M.D., Singh, S.P., De Sanctis, G.T., Eynott, P.R., Hahn, F.F., Campen, M.J., Mauderly, J.L. Toxicol. Sci. (2005) [Pubmed]
  8. DNA synthesis and Bcl-2 expression during development of mucous cell metaplasia in airway epithelium of rats exposed to LPS. Tesfaigzi, Y., Harris, J.F., Hotchkiss, J.A., Harkema, J.R. Am. J. Physiol. Lung Cell Mol. Physiol. (2004) [Pubmed]
  9. SPRR1B overexpression enhances entry of cells into the G0 phase of the cell cycle. Tesfaigzi, Y., Wright, P.S., Belinsky, S.A. Am. J. Physiol. Lung Cell Mol. Physiol. (2003) [Pubmed]
  10. Processes involved in the repair of injured airway epithelia. Tesfaigzi, Y. Arch. Immunol. Ther. Exp. (Warsz.) (2003) [Pubmed]
  11. Health effects of subchronic exposure to low levels of wood smoke in rats. Tesfaigzi, Y., Singh, S.P., Foster, J.E., Kubatko, J., Barr, E.B., Fine, P.M., McDonald, J.D., Hahn, F.F., Mauderly, J.L. Toxicol. Sci. (2002) [Pubmed]
  12. Bax is crucial for IFN-gamma-induced resolution of allergen-induced mucus cell metaplasia. Tesfaigzi, Y., Fischer, M.J., Daheshia, M., Green, F.H., De Sanctis, G.T., Wilder, J.A. J. Immunol. (2002) [Pubmed]
  13. Clinical and cellular effects of cytochrome P-450 modulators. Tesfaigzi, Y., Kluger, M., Kozak, W. Respir. Physiol (2001) [Pubmed]
  14. Bcl-2 mediates sex-specific differences in recovery of mice from LPS-induced signs of sickness independent of IL-6. Tesfaigzi, Y., Rudolph, K., Fischer, M.J., Conn, C.A. J. Appl. Physiol. (2001) [Pubmed]
  15. Bcl-2 in LPS- and allergen-induced hyperplastic mucous cells in airway epithelia of Brown Norway rats. Tesfaigzi, Y., Fischer, M.J., Martin, A.J., Seagrave, J. Am. J. Physiol. Lung Cell Mol. Physiol. (2000) [Pubmed]
 
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