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Driss Zoukhri

Department of Ophthalmology

Schepens Eye Research Institute

Harvard Medical School

20 Staniford Street

USA

[email]@*.eri.harvard.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Ophthalmology, Schepens Eye Research Institute, Harvard Medical School, 20 Staniford Street, USA. 2002
  • Department of Ophthalmology, Harvard Medical School, Boston, Massachusetts, USA. 1998 - 2001
  • Schepens Eye Research Institute, Boston, Massachusetts, USA. 1997 - 1998

References

  1. Role of proinflammatory cytokines in the impaired lacrimation associated with autoimmune xerophthalmia. Zoukhri, D., Hodges, R.R., Byon, D., Kublin, C.L. Invest. Ophthalmol. Vis. Sci. (2002) [Pubmed]
  2. Impaired neurotransmitter release from lacrimal and salivary gland nerves of a murine model of Sjögren's syndrome. Zoukhri, D., Kublin, C.L. Invest. Ophthalmol. Vis. Sci. (2001) [Pubmed]
  3. Cholinergic-induced Ca2+ elevation in rat lacrimal gland acini is negatively modulated by PKCdelta and PKCepsilon. Zoukhri, D., Hodges, R.R., Sergheraert, C., Dartt, D.A. Invest. Ophthalmol. Vis. Sci. (2000) [Pubmed]
  4. Lacrimal gland functions are differentially controlled by protein kinase C isoforms. Zoukhri, D., Hodges, R.R., Sergheraert, C., Dartt, D.A. Ann. N. Y. Acad. Sci. (1998) [Pubmed]
  5. Lacrimal gland innervation is not altered with the onset and progression of disease in a murine model of Sjögren's syndrome. Zoukhri, D., Hodges, R.R., Dartt, D.A. Clin. Immunol. Immunopathol. (1998) [Pubmed]
  6. Ca2+ signaling by cholinergic and alpha1-adrenergic agonists is up-regulated in lacrimal and submandibular glands in a murine model of Sjögren's syndrome. Zoukhri, D., Hodges, R.R., Rawe, I.M., Dartt, D.A. Clin. Immunol. Immunopathol. (1998) [Pubmed]
  7. Lacrimal gland PKC isoforms are differentially involved in agonist-induced protein secretion. Zoukhri, D., Hodges, R.R., Sergheraert, C., Toker, A., Dartt, D.A. Am. J. Physiol. (1997) [Pubmed]
 
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