S.S. Bolz
Physiologisches Institut
Ludwig Maximilians Universität
München
Germany
Name/email consistency: high
- Nitric oxide-induced decrease in calcium sensitivity of resistance arteries is attributable to activation of the myosin light chain phosphatase and antagonized by the RhoA/Rho kinase pathway. Bolz, S.S., Vogel, L., Sollinger, D., Derwand, R., de Wit, C., Loirand, G., Pohl, U. Circulation (2003)
- Sphingosine kinase modulates microvascular tone and myogenic responses through activation of RhoA/Rho kinase. Bolz, S.S., Vogel, L., Sollinger, D., Derwand, R., Boer, C., Pitson, S.M., Spiegel, S., Pohl, U. Circulation (2003)
- Highly effective non-viral gene transfer into vascular smooth muscle cells of cultured resistance arteries demonstrated by genetic inhibition of sphingosine-1-phosphate-induced vasoconstriction. Bolz, S.S., Pohl, U. J. Vasc. Res. (2003)
- Antisense oligonucleotides against cytochrome P450 2C8 attenuate EDHF-mediated Ca(2+) changes and dilation in isolated resistance arteries. Bolz, S.S., Fisslthaler, B., Pieperhoff, S., De Wit, C., Fleming, I., Busse, R., Pohl, U. FASEB J. (2000)
- Intact endothelial and smooth muscle function in small resistance arteries after 48 h in vessel culture. Bolz, S.S., Pieperhoff, S., De Wit, C., Pohl, U. Am. J. Physiol. Heart Circ. Physiol. (2000)
- Oxidized LDL increases the sensitivity of the contractile apparatus in isolated resistance arteries for Ca(2+) via a rho- and rho kinase-dependent mechanism. Bolz, S.S., Galle, J., Derwand, R., de Wit, C., Pohl, U. Circulation (2000)
