Alfred Ayala
Division of Surgical Research
Department of surgery
Aldrich 227
Brown University School Medicine/Rhode Island Hospital
USA
Name/email consistency: high
- Apoptosis in sepsis: mechanisms, clinical impact and potential therapeutic targets. Ayala, A., Perl, M., Venet, F., Lomas-Neira, J., Swan, R., Chung, C.S. Curr. Pharm. Des. (2008)
- SOCS-1 is a central mediator of steroid-increased thymocyte apoptosis and decreased survival following sepsis. Chung, C.S., Chen, Y., Grutkoski, P.S., Doughty, L., Ayala, A. Apoptosis (2007)
- CXCR2 inhibition suppresses hemorrhage-induced priming for acute lung injury in mice. Lomas-Neira, J.L., Chung, C.S., Grutkoski, P.S., Miller, E.J., Ayala, A. J. Leukoc. Biol. (2004)
- Pathological aspects of apoptosis in severe sepsis and shock?. Ayala, A., Lomas, J.L., Grutkoski, P.S., Chung, C.S. Int. J. Biochem. Cell Biol. (2003)
- Mechanisms of immune resolution. Ayala, A., Chung, C.S., Grutkoski, P.S., Song, G.Y. Crit. Care Med. (2003)
- Fas-ligand mediated apoptosis in severe sepsis and shock. Ayala, A., Lomas, J.L., Grutkoski, P.S., Chung, S. Scand. J. Infect. Dis. (2003)
- Shock-induced neutrophil mediated priming for acute lung injury in mice: divergent effects of TLR-4 and TLR-4/FasL deficiency. Ayala, A., Chung, C.S., Lomas, J.L., Song, G.Y., Doughty, L.A., Gregory, S.H., Cioffi, W.G., LeBlanc, B.W., Reichner, J., Simms, H.H., Grutkoski, P.S. Am. J. Pathol. (2002)
- IL-10 mediation of activation-induced TH1 cell apoptosis and lymphoid dysfunction in polymicrobial sepsis. Ayala, A., Chung, C.S., Song, G.Y., Chaudry, I.H. Cytokine (2001)
- Immune depression in polymicrobial sepsis: the role of necrotic (injured) tissue and endotoxin. Ayala, A., Song, G.Y., Chung, C.S., Redmond, K.M., Chaudry, I.H. Crit. Care Med. (2000)
