C.C. Chen
Institute of Pharmacology
College of Medicine
National Taiwan University
Taipei
Name/email consistency: high
- Signal transduction pathways of inflammatory gene expressions and therapeutic implications. Chen, C.C. Curr. Pharm. Des. (2006)
- Flavonoids inhibit tumor necrosis factor-alpha-induced up-regulation of intercellular adhesion molecule-1 (ICAM-1) in respiratory epithelial cells through activator protein-1 and nuclear factor-kappaB: structure-activity relationships. Chen, C.C., Chow, M.P., Huang, W.C., Lin, Y.C., Chang, Y.J. Mol. Pharmacol. (2004)
- Tumor necrosis factor alpha-induced activation of downstream NF-kappaB site of the promoter mediates epithelial ICAM-1 expression and monocyte adhesion. Involvement of PKCalpha, tyrosine kinase, and IKK2, but not MAPKs, pathway. Chen, C., Chou, C., Sun, Y., Huang, W. Cell. Signal. (2001)
- Conjugated polyhydroxybenzene derivatives block tumor necrosis factor-alpha-mediated nuclear factor-kappaB activation and cyclooxygenase-2 gene transcription by targeting IkappaB kinase activity. Chen, C.C., Chiu, K.T., Chan, S.T., Chern, J.W. Mol. Pharmacol. (2001)
- TNF-alpha-induced cyclooxygenase-2 expression in human lung epithelial cells: involvement of the phospholipase C-gamma 2, protein kinase C-alpha, tyrosine kinase, NF-kappa B-inducing kinase, and I-kappa B kinase 1/2 pathway. Chen, C.C., Sun, Y.T., Chen, J.J., Chiu, K.T. J. Immunol. (2000)
- Protein kinase calpha but not p44/42 mitogen-activated protein kinase, p38, or c-Jun NH(2)-terminal kinase is required for intercellular adhesion molecule-1 expression mediated by interleukin-1beta: involvement of sequential activation of tyrosine kinase, nuclear factor-kappaB-inducing kinase, and IkappaB kinase 2. Chen, C.C., Chen, J.J., Chou, C.Y. Mol. Pharmacol. (2000)
- p38 but not p44/42 mitogen-activated protein kinase is required for nitric oxide synthase induction mediated by lipopolysaccharide in RAW 264.7 macrophages. Chen, C.C., Wang, J.K. Mol. Pharmacol. (1999)
- Antisense oligonucleotides targeting protein kinase C-alpha, -beta I, or -delta but not -eta inhibit lipopolysaccharide-induced nitric oxide synthase expression in RAW 264.7 macrophages: involvement of a nuclear factor kappa B-dependent mechanism. Chen, C.C., Wang, J.K., Lin, S.B. J. Immunol. (1998)
