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David E. J. Jones

Institute of Cellular Medicine

Newcastle University

Newcastle-upon-Tyne

UK

[email]@ncl.ac.uk

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Institute of Cellular Medicine, Newcastle University, Newcastle-upon-Tyne, UK. 2010
  • Centre for Liver Research, 4th Floor William Leech Building, The Medical School, University of Newcastle, UK. 2002 - 2008
  • Liver Research Group, Institute of Cellular Medicine, The Medical School, Newcastle University, United Kingdom. 2008
  • Centre for Liver Research, University of Newcastle, Framlington Place, Newcastle-upon-Tyne, UK. 1999 - 2007
  • Centre for Liver Research, University of Newcastle, School of Clinical Medical Sciences, 4th Floor William Leech Building, UK. 2003 - 2006
  • School of Clinical Medical Sciences, Medical School, University of Newcastle, Framlington Place, UK. 2004
  • Centre for Liver Research, University of Newcastle, UK. 1999 - 2003

References

  1. The independent effects of fatigue and UDCA therapy on mortality in primary biliary cirrhosis: results of a 9 year follow-up. Jones, D.E., Al-Rifai, A., Frith, J., Patanwala, I., Newton, J.L. J. Hepatol. (2010) [Pubmed]
  2. Pathogenesis of primary biliary cirrhosis. Jones, D.E. Postgrad. Med. J (2008) [Pubmed]
  3. Pathogenesis of primary biliary cirrhosis. Jones, D.E. Clin. Liver. Dis (2008) [Pubmed]
  4. An open study of modafinil for the treatment of daytime somnolence and fatigue in primary biliary cirrhosis. Jones, D.E., Newton, J.L. Aliment. Pharmacol. Ther. (2007) [Pubmed]
  5. Pathogenesis of primary biliary cirrhosis. Jones, D.E. Gut (2007) [Pubmed]
  6. Four year follow up of fatigue in a geographically defined primary biliary cirrhosis patient cohort. Jones, D.E., Bhala, N., Burt, J., Goldblatt, J., Prince, M., Newton, J.L. Gut (2006) [Pubmed]
  7. Primary biliary cirrhosis. Jones, D.E. Autoimmunity (2004) [Pubmed]
  8. Addison's other disease: primary biliary cirrhosis as a model autoimmune disease. Jones, D.E. Clin. Med (2003) [Pubmed]
  9. Pathogenesis of primary biliary cirrhosis. Jones, D.E. J. Hepatol. (2003) [Pubmed]
  10. Genetic factors in the pathogenesis of primary biliary cirrhosis. Jones, D.E., Donaldson, P.T. Clin. Liver. Dis (2003) [Pubmed]
  11. Bacterial motif DNA as an adjuvant for the breakdown of immune self-tolerance to pyruvate dehydrogenase complex. Jones, D.E., Palmer, J.M., Burt, A.D., Walker, C., Robe, A.J., Kirby, J.A. Hepatology (2002) [Pubmed]
  12. Oral tolerisation to pyruvate dehydrogenase complex as a potential therapy for primary biliary cirrhosis. Jones, D.E., Palmer, J.M., Robe, A., Kirby, J.A. Autoimmunity (2002) [Pubmed]
  13. Experimental autoimmune cholangitis: a mouse model of immune-mediated cholangiopathy. Jones, D.E., Palmer, J.M., Kirby, J.A., De Cruz, D.J., McCaughan, G.W., Sedgwick, J.D., Yeaman, S.J., Burt, A.D., Bassendine, M.F. Liver (2000) [Pubmed]
  14. Autoantigens in primary biliary cirrhosis. Jones, D.E. J. Clin. Pathol. (2000) [Pubmed]
  15. Familial primary biliary cirrhosis reassessed: a geographically-based population study. Jones, D.E., Watt, F.E., Metcalf, J.V., Bassendine, M.F., James, O.F. J. Hepatol. (1999) [Pubmed]
  16. Breakdown of tolerance to pyruvate dehydrogenase complex in experimental autoimmune cholangitis: a mouse model of primary biliary cirrhosis. Jones, D.E., Palmer, J.M., Yeaman, S.J., Kirby, J.A., Bassendine, M.F. Hepatology (1999) [Pubmed]
 
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