Daniel J. J. Carr
Department of Microbiology and Immunology
University of Oklahoma Health Sciences Center
Oklahoma City
OK 73104
USA
Name/email consistency: high
- Delivery of Interferon-gamma by an adenovirus vector blocks herpes simplex virus Type 1 reactivation in vitro and in vivo independent of RNase L and double-stranded RNA-dependent protein kinase pathways. Carr, D.J., Austin, B.A., Halford, W.P., Stuart, P.M. J. Neuroimmunol. (2009)
- An increase in herpes simplex virus type 1 in the anterior segment of the eye is linked to a deficiency in NK cell infiltration in mice deficient in CXCR3. Carr, D.J., Wuest, T., Ash, J. J. Interferon Cytokine Res. (2008)
- Abnormal immune response of CCR5-deficient mice to ocular infection with herpes simplex virus type 1. Carr, D.J., Ash, J., Lane, T.E., Kuziel, W.A. J. Gen. Virol. (2006)
- Herpes simplex virus and the chemokines that mediate the inflammation. Carr, D.J., Tomanek, L. Curr. Top. Microbiol. Immunol. (2006)
- The lack of RNA-dependent protein kinase enhances susceptibility of mice to genital herpes simplex virus type 2 infection. Carr, D.J., Wuest, T., Tomanek, L., Silverman, R.H., Williams, B.R. Immunology (2006)
- Herpes simplex virus type 1 induction of chemokine production is unrelated to viral load in the cornea but not in the nervous system. Carr, D.J., Campbell, I.L. Viral Immunol. (2006)
- RNA-dependent protein kinase is required for alpha-1 interferon transgene-induced resistance to genital herpes simplex virus type 2. Carr, D.J., Tomanek, L., Silverman, R.H., Campbell, I.L., Williams, B.R. J. Virol. (2005)
- Effect of anti-CXCL10 monoclonal antibody on herpes simplex virus type 1 keratitis and retinal infection. Carr, D.J., Chodosh, J., Ash, J., Lane, T.E. J. Virol. (2003)
- Interferon-beta suppresses herpes simplex virus type 1 replication in trigeminal ganglion cells through an RNase L-dependent pathway. Carr, D.J., Al-khatib, K., James, C.M., Silverman, R. J. Neuroimmunol. (2003)
- The antiviral efficacy of the murine alpha-1 interferon transgene against ocular herpes simplex virus type 1 requires the presence of CD4(+), alpha/beta T-cell receptor-positive T lymphocytes with the capacity to produce gamma interferon. Carr, D.J., Noisakran, S. J. Virol. (2002)
- The immune response to ocular herpes simplex virus type 1 infection. Carr, D.J., Härle, P., Gebhardt, B.M. Exp. Biol. Med. (Maywood) (2001)
