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D. Sanglard

Institut de Microbiologie

Centre Hospitalier Universitaire Vaudois

Lausanne

Switzerland

[email]@*.unil.ch

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Affiliation

Institut de Microbiologie, Centre Hospitalier Universitaire Vaudois, Lausanne, Switzerland. 1996 - 1998

References

Multiple resistance mechanisms to azole antifungals in yeast clinical isolates. Sanglard, D., Ischer, F., Calabrese, D., Micheli, M., Bille, J. Drug Resist. Updat. (1998) [Pubmed]
Amino acid substitutions in the cytochrome P-450 lanosterol 14alpha-demethylase (CYP51A1) from azole-resistant Candida albicans clinical isolates contribute to resistance to azole antifungal agents. Sanglard, D., Ischer, F., Koymans, L., Bille, J. Antimicrob. Agents Chemother. (1998) [Pubmed]
Cloning of Candida albicans genes conferring resistance to azole antifungal agents: characterization of CDR2, a new multidrug ABC transporter gene. Sanglard, D., Ischer, F., Monod, M., Bille, J. Microbiology (Reading, Engl.) (1997) [Pubmed]
A triple deletion of the secreted aspartyl proteinase genes SAP4, SAP5, and SAP6 of Candida albicans causes attenuated virulence. Sanglard, D., Hube, B., Monod, M., Odds, F.C., Gow, N.A. Infect. Immun. (1997) [Pubmed]
Susceptibilities of Candida albicans multidrug transporter mutants to various antifungal agents and other metabolic inhibitors. Sanglard, D., Ischer, F., Monod, M., Bille, J. Antimicrob. Agents Chemother. (1996) [Pubmed]