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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

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Douglas R. Yingst

Department of Physiology

Wayne State University School of Medicine

Detroit

MI 48201

USA

[email]@med.wayne.edu

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Department of Physiology, Wayne State University School of Medicine, Detroit, MI 48201, USA. 2000 - 2009

References

Decreased renal perfusion rapidly increases plasma membrane Na-K-ATPase in rat cortex by an angiotensin II-dependent mechanism. Yingst, D.R., Araghi, A., Doci, T.M., Mattingly, R., Beierwaltes, W.H. Am. J. Physiol. Renal Physiol. (2009) [Pubmed]
Angiotensin II stimulates elution of Na-K-ATPase from a digoxin-affinity column by increasing the kinetic response to ligands that trigger the decay of E2-P. Yingst, D.R., Doci, T.M., Massey, K.J., Rossi, N.F., Rucker, E., Mattingly, R.R. Am. J. Physiol. Renal Physiol. (2008) [Pubmed]
Angiotensin II directly stimulates activity and alters the phosphorylation of Na-K-ATPase in rat proximal tubule with a rapid time course. Yingst, D.R., Massey, K.J., Rossi, N.F., Mohanty, M.J., Mattingly, R.R. Am. J. Physiol. Renal Physiol. (2004) [Pubmed]
Effects of extracellular calcium and potassium on the sodium pump of rat adrenal glomerulosa cells. Yingst, D.R., Davis, J., Schiebinger, R. Am. J. Physiol., Cell Physiol. (2001) [Pubmed]
Inhibitors of tyrosine phosphatases block angiotensin II inhibition of Na(+) pump. Yingst, D.R., Davis, J., Schiebinger, R. Eur. J. Pharmacol. (2000) [Pubmed]

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