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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Gary B. Huffnagle

Address correspondence to Gary B. Huffnagle

[email]@umich.edu

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Affiliations

  • Address correspondence to Gary B. Huffnagle. 2012
  • Department of Internal Medicine, Division of Pulmonary and Critical Care Medicine, University of Michigan, Ann Arbor, USA. 2003
  • Department of Internal Medicine, University of Michigan Medical Center, Ann Arbor 48109, USA. 1997 - 2000

References

  1. Interleukin-17 Drives Pulmonary Eosinophilia following Repeated Exposure to Aspergillus fumigatus Conidia. Murdock, B.J., Falkowski, N.R., Shreiner, A.B., Akha, A.A., McDonald, R.A., White, E.S., Toews, G.B., Huffnagle, G.B. Infect. Immun. (2012) [Pubmed]
  2. Innate and adaptive determinants of host susceptibility to medically important fungi. Huffnagle, G.B., Deepe, G.S. Curr. Opin. Microbiol. (2003) [Pubmed]
  3. Leukocyte recruitment during pulmonary Cryptococcus neoformans infection. Huffnagle, G.B., Traynor, T.R., McDonald, R.A., Olszewski, M.A., Lindell, D.M., Herring, A.C., Toews, G.B. Immunopharmacology (2000) [Pubmed]
  4. Cutting edge: Role of C-C chemokine receptor 5 in organ-specific and innate immunity to Cryptococcus neoformans. Huffnagle, G.B., McNeil, L.K., McDonald, R.A., Murphy, J.W., Toews, G.B., Maeda, N., Kuziel, W.A. J. Immunol. (1999) [Pubmed]
  5. IL-5 is required for eosinophil recruitment, crystal deposition, and mononuclear cell recruitment during a pulmonary Cryptococcus neoformans infection in genetically susceptible mice (C57BL/6). Huffnagle, G.B., Boyd, M.B., Street, N.E., Lipscomb, M.F. J. Immunol. (1998) [Pubmed]
  6. Macrophage inflammatory protein-1alpha (MIP-1alpha) is required for the efferent phase of pulmonary cell-mediated immunity to a Cryptococcus neoformans infection. Huffnagle, G.B., Strieter, R.M., McNeil, L.K., McDonald, R.A., Burdick, M.D., Kunkel, S.L., Toews, G.B. J. Immunol. (1997) [Pubmed]
 
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