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John N. Wood

Molecular Nociception Group

Wolfson Institute for Biomedical Research (WIBR)

Cruciform Building

University College London (UCL)

UK

[email]@ucl.ac.uk

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Molecular Nociception Group, Wolfson Institute for Biomedical Research (WIBR), Cruciform Building, University College London (UCL), UK. 2004 - 2011
  • Department of Biology, University College, London WC1E 6BT, United Kingdom. 2000 - 2004

References

  1. Temporal control of gene deletion in sensory ganglia using a tamoxifen-inducible Advillin-Cre-ERT2 recombinase mouse. Lau, J., Minett, M.S., Zhao, J., Dennehy, U., Wang, F., Wood, J.N., Bogdanov, Y.D. Mol. Pain (2011) [Pubmed]
  2. Pain as a channelopathy. Raouf, R., Quick, K., Wood, J.N. J. Clin. Invest. (2010) [Pubmed]
  3. Ion channels in analgesia research. Wood, J.N. Handb. Exp. Pharmacol (2007) [Pubmed]
  4. Voltage-gated sodium channel blockers; target validation and therapeutic potential. Wood, J.N., Boorman, J. Curr. Top. Med. Chem (2005) [Pubmed]
  5. Recent advances in understanding molecular mechanisms of primary afferent activation. Wood, J.N. Gut (2004) [Pubmed]
  6. Voltage-gated sodium channels and pain pathways. Wood, J.N., Boorman, J.P., Okuse, K., Baker, M.D. J. Neurobiol. (2004) [Pubmed]
  7. Voltage-gated sodium channels. Wood, J.N., Baker, M. Curr. Opin. Pharmacol (2001) [Pubmed]
  8. Pathobiology of visceral pain: molecular mechanisms and therapeutic implications. II. Genetic approaches to pain therapy. Wood, J.N. Am. J. Physiol. Gastrointest. Liver Physiol. (2000) [Pubmed]
  9. Molecules that specify modality: mechanisms of nociception. Wood, J.N., Heath, M.J. J. Pain (2000) [Pubmed]
 
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