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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

James F. Dillman

Applied Pharmacology Branch

U.S. Army Medical Research Institute of Chemical Defense

Aberdeen Proving Ground

MD 21010-5400

USA

[email]@*.*.army.mil

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Applied Pharmacology Branch, U.S. Army Medical Research Institute of Chemical Defense, Aberdeen Proving Ground, MD 21010-5400, USA. 2003 - 2005

References

  1. Genomic analysis of rodent pulmonary tissue following bis-(2-chloroethyl) sulfide exposure. Dillman, J.F., Phillips, C.S., Dorsch, L.M., Croxton, M.D., Hege, A.I., Sylvester, A.J., Moran, T.S., Sciuto, A.M. Chem. Res. Toxicol. (2005) [Pubmed]
  2. Comparison of non-human primate and human whole blood tissue gene expression profiles. Dillman, J.F., Phillips, C.S. Toxicol. Sci. (2005) [Pubmed]
  3. An inhibitor of p38 MAP kinase downregulates cytokine release induced by sulfur mustard exposure in human epidermal keratinocytes. Dillman, J.F., McGary, K.L., Schlager, J.J. Toxicol. In. Vitro (2004) [Pubmed]
  4. Sulfur mustard induces the formation of keratin aggregates in human epidermal keratinocytes. Dillman, J.F., McGary, K.L., Schlager, J.J. Toxicol. Appl. Pharmacol. (2003) [Pubmed]
 
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