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John H. Richburg

Division of Pharmacology and Toxicology

University of Texas College of Pharmacy

Austin 78712-1074

USA

[email]@*.utexas.edu

Name/email consistency: high

Affiliations

Division of Pharmacology and Toxicology, University of Texas College of Pharmacy, Austin 78712-1074, USA. 2000 - 2003
College of Pharmacy, The University of Texas at Austin, Austin, Texas, USA. 1999 - 2000

References

Fas- or FasL-deficient mice display an increased sensitivity to nitrobenzene-induced testicular germ cell apoptosis. Richburg, J.H., Nañez, A. Toxicol. Lett. (2003) [Pubmed]
Defining the cellular and molecular mechanisms of toxicant action in the testis. Richburg, J.H., Johnson, K.J., Schoenfeld, H.A., Meistrich, M.L., Dix, D.J. Toxicol. Lett. (2002) [Pubmed]
Sensitivity of testicular germ cells to toxicant-induced apoptosis in gld mice that express a nonfunctional form of Fas ligand. Richburg, J.H., Nañez, A., Williams, L.R., Embree, M.E., Boekelheide, K. Endocrinology (2000) [Pubmed]
The relevance of spontaneous- and chemically-induced alterations in testicular germ cell apoptosis to toxicology. Richburg, J.H. Toxicol. Lett. (2000) [Pubmed]
Participation of the Fas-signaling system in the initiation of germ cell apoptosis in young rat testes after exposure to mono-(2-ethylhexyl) phthalate. Richburg, J.H., Nañez, A., Gao, H. Toxicol. Appl. Pharmacol. (1999) [Pubmed]