Uwe Kirchhefer
Institut für Pharmakologie und Toxikologie
Universitätsklinikum Münster
Domagkstr. 12
48149 Münster
Germany
Name/email consistency: high
- The human CASQ2 mutation K206N is associated with hyperglycosylation and altered cellular calcium handling. Kirchhefer, U., Wehrmeister, D., Postma, A.V., Pohlentz, G., Mormann, M., Kucerova, D., Müller, F.U., Schmitz, W., Schulze-Bahr, E., Wilde, A.A., Neumann, J. J. Mol. Cell. Cardiol. (2010)
- Triadin is a critical determinant of cellular Ca cycling and contractility in the heart. Kirchhefer, U., Klimas, J., Baba, H.A., Buchwalow, I.B., Fabritz, L., Hüls, M., Matus, M., Müller, F.U., Schmitz, W., Neumann, J. Am. J. Physiol. Heart Circ. Physiol. (2007)
- Transgenic triadin 1 overexpression alters SR Ca2+ handling and leads to a blunted contractile response to beta-adrenergic agonists. Kirchhefer, U., Jones, L.R., Begrow, F., Boknik, P., Hein, L., Lohse, M.J., Riemann, B., Schmitz, W., Stypmann, J., Neumann, J. Cardiovasc. Res. (2004)
- Age-dependent biochemical and contractile properties in atrium of transgenic mice overexpressing junctin. Kirchhefer, U., Baba, H.A., Hanske, G., Jones, L.R., Kirchhof, P., Schmitz, W., Neumann, J. Am. J. Physiol. Heart Circ. Physiol. (2004)
- Altered function in atrium of transgenic mice overexpressing triadin 1. Kirchhefer, U., Baba, H.A., Kobayashi, Y.M., Jones, L.R., Schmitz, W., Neumann, J. Am. J. Physiol. Heart Circ. Physiol. (2002)
- Cardiac hypertrophy and impaired relaxation in transgenic mice overexpressing triadin 1. Kirchhefer, U., Neumann, J., Baba, H.A., Begrow, F., Kobayashi, Y.M., Reinke, U., Schmitz, W., Jones, L.R. J. Biol. Chem. (2001)
