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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Marja Pertovaara

Department of Internal Medicine

Section of Rheumatology

Tampere University Hospital

P.O. Box 2000

Finland

[email]@pshp.fi

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Internal Medicine, Section of Rheumatology, Tampere University Hospital, P.O. Box 2000, Finland. 2004 - 2006
  • Tampere University Hospital, Finland. 2005

References

  1. Th2 cytokine genotypes are associated with a milder form of primary Sjogren's syndrome. Pertovaara, M., Antonen, J., Hurme, M. Ann. Rheum. Dis. (2006) [Pubmed]
  2. Mechanisms dependent on tryptophan catabolism regulate immune responses in primary Sjögren's syndrome. Pertovaara, M., Raitala, A., Uusitalo, H., Pukander, J., Helin, H., Oja, S.S., Hurme, M. Clin. Exp. Immunol. (2005) [Pubmed]
  3. Presence of apolipoprotein E epsilon4 allele predisposes to early onset of primary Sjogren's syndrome. Pertovaara, M., Lehtimäki, T., Rontu, R., Antonen, J., Pasternack, A., Hurme, M. Rheumatology. (Oxford) (2004) [Pubmed]
  4. Long-term follow-up study of pulmonary findings in patients with primary Sjögren's syndrome. Pertovaara, M., Korpela, M., Saarelainen, S., Laitinen, J., Järvenpää, R., Laippala, P., Lahdensuo, A. Scand. J. Rheumatol. (2004) [Pubmed]
  5. Immunoglobulin KM and GM gene polymorphisms modify the clinical presentation of primary Sjögren's syndrome. Pertovaara, M., Hurme, M., Antonen, J., Pasternack, A., Pandey, J.P. J. Rheumatol. (2004) [Pubmed]
 
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