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Nicholas Michael Morton

Corresponding author: Nicholas M. Morton

[email]@ed.ac.uk

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Corresponding author: Nicholas M. Morton. 2012
  • Molecular Metabolism Group, BHF-University Centre for Cardiovascular Sciences, Queens Medical Research Institute, Univ. of Edinburgh. 2010 - 2011
  • Molecular Endocrinology, Molecular Medicine Centre, University of Edinburgh, Western General Hospital, United Kingdom. 2001 - 2005

References

  1. Optimal Elevation of β-Cell 11β-Hydroxysteroid Dehydrogenase Type 1 Is a Compensatory Mechanism That Prevents High-Fat Diet-Induced β-Cell Failure. Turban, S., Liu, X., Ramage, L., Webster, S.P., Walker, B.R., Dunbar, D.R., Mullins, J.J., Seckl, J.R., Morton, N.M. Diabetes (2012) [Pubmed]
  2. Dietary manipulation reveals an unexpected inverse relationship between fat mass and adipose 11{beta}-hydroxysteroid dehydrogenase type 1. Man, T.Y., Michailidou, Z., Gokcel, A., Ramage, L., Chapman, K.E., Kenyon, C.J., Seckl, J.R., Morton, N.M. Am. J. Physiol. Endocrinol. Metab. (2011) [Pubmed]
  3. Obesity and corticosteroids: 11beta-hydroxysteroid type 1 as a cause and therapeutic target in metabolic disease. Morton, N.M. Mol. Cell. Endocrinol. (2010) [Pubmed]
  4. A polygenic model of the metabolic syndrome with reduced circulating and intra-adipose glucocorticoid action. Morton, N.M., Densmore, V., Wamil, M., Ramage, L., Nichol, K., Bünger, L., Seckl, J.R., Kenyon, C.J. Diabetes (2005) [Pubmed]
  5. Down-regulation of adipose 11beta-hydroxysteroid dehydrogenase type 1 by high-fat feeding in mice: a potential adaptive mechanism counteracting metabolic disease. Morton, N.M., Ramage, L., Seckl, J.R. Endocrinology (2004) [Pubmed]
  6. Novel adipose tissue-mediated resistance to diet-induced visceral obesity in 11 beta-hydroxysteroid dehydrogenase type 1-deficient mice. Morton, N.M., Paterson, J.M., Masuzaki, H., Holmes, M.C., Staels, B., Fievet, C., Walker, B.R., Flier, J.S., Mullins, J.J., Seckl, J.R. Diabetes (2004) [Pubmed]
  7. Improved lipid and lipoprotein profile, hepatic insulin sensitivity, and glucose tolerance in 11beta-hydroxysteroid dehydrogenase type 1 null mice. Morton, N.M., Holmes, M.C., Fiévet, C., Staels, B., Tailleux, A., Mullins, J.J., Seckl, J.R. J. Biol. Chem. (2001) [Pubmed]
 
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