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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Halina Offner

DrMed

Neuroimmunology Research

R&D-31

Portland Veterans Affairs Medical Center

[email]@ohsu.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • DrMed, Neuroimmunology Research, R&D-31, Portland Veterans Affairs Medical Center. 2011
  • Neuroimmunology Research, Veterans Affairs Medical Center, 3710 SW US Veterans' Hospital Rd., Portland, United States. 2004 - 2011
  • Department of Neurology, Oregon Health and Science University, Neuroimmunology Research, Veterans Affairs Medical Center, USA. 2004
  • Department of Neurology, Oregon Health and Science University, 97201, Portland, USA. 2002

References

  1. Programmed death-1 pathway limits central nervous system inflammation and neurologic deficits in murine experimental stroke. Ren, X., Akiyoshi, K., Vandenbark, A.A., Hurn, P.D., Offner, H. Stroke (2011) [Pubmed]
  2. RTL therapy for multiple sclerosis: a Phase I clinical study. Offner, H., Sinha, S., Burrows, G.G., Ferro, A.J., Vandenbark, A.A. J. Neuroimmunol. (2011) [Pubmed]
  3. Effect of experimental stroke on peripheral immunity: CNS ischemia induces profound immunosuppression. Offner, H., Vandenbark, A.A., Hurn, P.D. Neuroscience (2009) [Pubmed]
  4. Recombinant T cell receptor ligands: immunomodulatory, neuroprotective and neuroregenerative effects suggest application as therapy for multiple sclerosis. Offner, H., Sinha, S., Wang, C., Burrows, G.G., Vandenbark, A.A. Rev. Neurosci (2008) [Pubmed]
  5. Splenic atrophy in experimental stroke is accompanied by increased regulatory T cells and circulating macrophages. Offner, H., Subramanian, S., Parker, S.M., Wang, C., Afentoulis, M.E., Lewis, A., Vandenbark, A.A., Hurn, P.D. J. Immunol. (2006) [Pubmed]
  6. Experimental stroke induces massive, rapid activation of the peripheral immune system. Offner, H., Subramanian, S., Parker, S.M., Afentoulis, M.E., Vandenbark, A.A., Hurn, P.D. J. Cereb. Blood Flow Metab. (2006) [Pubmed]
  7. A potential role for estrogen in experimental autoimmune encephalomyelitis and multiple sclerosis. Offner, H., Polanczyk, M. Ann. N. Y. Acad. Sci. (2006) [Pubmed]
  8. Treatment of passive experimental autoimmune encephalomyelitis in SJL mice with a recombinant TCR ligand induces IL-13 and prevents axonal injury. Offner, H., Subramanian, S., Wang, C., Afentoulis, M., Vandenbark, A.A., Huan, J., Burrows, G.G. J. Immunol. (2005) [Pubmed]
  9. Congruent effects of estrogen and T-cell receptor peptide therapy on regulatory T cells in EAE and MS. Offner, H., Vandenbark, A.A. Int. Rev. Immunol. (2005) [Pubmed]
  10. A synthetic androstene analogue inhibits collagen-induced arthritis in the mouse. Offner, H., Zamora, A., Subramanian, S., Polanczyk, M., Krogstad, A., Auci, D.L., Morgan, E.E., Reading, C.L. Clin. Immunol. (2004) [Pubmed]
  11. Neuroimmunoprotective effects of estrogen and derivatives in experimental autoimmune encephalomyelitis: therapeutic implications for multiple sclerosis. Offner, H. J. Neurosci. Res. (2004) [Pubmed]
  12. A synthetic androstene derivative and a natural androstene metabolite inhibit relapsing-remitting EAE. Offner, H., Zamora, A., Drought, H., Matejuk, A., Auci, D.L., Morgan, E.E., Vandenbark, A.A., Reading, C.L. J. Neuroimmunol. (2002) [Pubmed]
 
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