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Ozlem Equils

Department of Pediatrics

Burns and Allen Research Institute

Cedars-Sinai Medical Center

David Geffen School of Medicine at University of California Los Angeles

United States of America

[email]@cshs.org

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Pediatrics, Burns and Allen Research Institute, Cedars-Sinai Medical Center, David Geffen School of Medicine at University of California Los Angeles, United States of America. 2010
  • Burns and Allen Research Institute, Cedars-Sinai Medical Center, Los Angeles, CA 90048, USA. 2009
  • Division of Pediatric Infectious Diseases, Ahmanson Department of Pediatrics, Steven Spielberg Pediatric Research Center, Burns and Allen Research Institute, USA. 2001 - 2006
  • Department of Pediatrics, Cedars-Sinai Medical Center David Geffen School of Medicine at UCLA, Los Angeles, CA 90048, USA. 2005 - 2006
  • Department of Pediatrics, Cedars-Sinai Medical Center, 8700 Beverly Blvd., Room 4220, USA. 2004

References

  1. A computer simulation of progesterone and Cox2 inhibitor treatment for preterm labor. Equils, O., Nambiar, P., Hobel, C.J., Smith, R., Simmons, C.F., Vali, S. PLoS. ONE (2010) [Pubmed]
  2. Pretreatment with pancaspase inhibitor (Z-VAD-FMK) delays but does not prevent intraperitoneal heat-killed group B Streptococcus-induced preterm delivery in a pregnant mouse model. Equils, O., Moffatt-Blue, C., Ishikawa, T.O., Simmons, C.F., Ilievski, V., Hirsch, E. Infect. Dis. Obstet. Gynecol (2009) [Pubmed]
  3. Repeated lipopolysaccharide (LPS) exposure inhibits HIV replication in primary human macrophages. Equils, O., Salehi, K.K., Cornataeanu, R., Lu, D., Singh, S., Whittaker, K., Baldwin, G.C. Microbes Infect. (2006) [Pubmed]
  4. 1,25-Dihydroxyvitamin D inhibits lipopolysaccharide-induced immune activation in human endothelial cells. Equils, O., Naiki, Y., Shapiro, A.M., Michelsen, K., Lu, D., Adams, J., Jordan, S. Clin. Exp. Immunol. (2006) [Pubmed]
  5. Chlamydia heat shock protein 60 induces trophoblast apoptosis through TLR4. Equils, O., Lu, D., Gatter, M., Witkin, S.S., Bertolotto, C., Arditi, M., McGregor, J.A., Simmons, C.F., Hobel, C.J. J. Immunol. (2006) [Pubmed]
  6. Intra-uterine growth restriction downregulates the hepatic toll like receptor-4 expression and function. Equils, O., Singh, S., Karaburun, S., Lu, D., Thamotharan, M., Devaskar, S.U. Clin. Dev. Immunol. (2005) [Pubmed]
  7. Rac1 and Toll-IL-1 receptor domain-containing adapter protein mediate Toll-like receptor 4 induction of HIV-long terminal repeat. Equils, O., Madak, Z., Liu, C., Michelsen, K.S., Bulut, Y., Lu, D. J. Immunol. (2004) [Pubmed]
  8. Human immunodeficiency virus type 1 protease inhibitors block toll-like receptor 2 (TLR2)- and TLR4-Induced NF-kappaB activation. Equils, O., Shapiro, A., Madak, Z., Liu, C., Lu, D. Antimicrob. Agents Chemother. (2004) [Pubmed]
  9. Toll-like receptor 2 (TLR2) and TLR9 signaling results in HIV-long terminal repeat trans-activation and HIV replication in HIV-1 transgenic mouse spleen cells: implications of simultaneous activation of TLRs on HIV replication. Equils, O., Schito, M.L., Karahashi, H., Madak, Z., Yarali, A., Michelsen, K.S., Sher, A., Arditi, M. J. Immunol. (2003) [Pubmed]
  10. Bacterial lipopolysaccharide activates HIV long terminal repeat through Toll-like receptor 4. Equils, O., Faure, E., Thomas, L., Bulut, Y., Trushin, S., Arditi, M. J. Immunol. (2001) [Pubmed]
 
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