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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Patrick J. Pollard

Molecular and Population Genetics Laboratory

Histopathology Department

London Research Institute

Cancer Research UK

United Kingdom

[email]@cancer.org.uk

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Molecular and Population Genetics Laboratory, Histopathology Department, London Research Institute, Cancer Research UK, United Kingdom. 2007
  • Molecular and Population Genetics Laboratory, Cancer Research UK, Lincoln's Inn Fields, London, UK. 2003 - 2005

References

  1. Targeted inactivation of fh1 causes proliferative renal cyst development and activation of the hypoxia pathway. Pollard, P.J., Spencer-Dene, B., Shukla, D., Howarth, K., Nye, E., El-Bahrawy, M., Deheragoda, M., Joannou, M., McDonald, S., Martin, A., Igarashi, P., Varsani-Brown, S., Rosewell, I., Poulsom, R., Maxwell, P., Stamp, G.W., Tomlinson, I.P. Cancer. Cell (2007) [Pubmed]
  2. Evidence of increased microvessel density and activation of the hypoxia pathway in tumours from the hereditary leiomyomatosis and renal cell cancer syndrome. Pollard, P., Wortham, N., Barclay, E., Alam, A., Elia, G., Manek, S., Poulsom, R., Tomlinson, I. J. Pathol. (2005) [Pubmed]
  3. The TCA cycle and tumorigenesis: the examples of fumarate hydratase and succinate dehydrogenase. Pollard, P.J., Wortham, N.C., Tomlinson, I.P. Ann. Med. (2003) [Pubmed]
 
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