Peter E. Light
Department of Pharmacology
Alberta Diabetes Institute
Faculty of Medicine and Dentistry
1-005 Li Ka Shing Centre for Health Research Innovation
Canada
Name/email consistency: high
- Late sodium current inhibition alone with ranolazine is sufficient to reduce ischemia- and cardiac glycoside-induced calcium overload and contractile dysfunction mediated by reverse-mode sodium/calcium exchange. Soliman, D., Wang, L., Hamming, K.S., Yang, W., Fatehi, M., Carter, C.C., Clanachan, A.S., Light, P.E. J. Pharmacol. Exp. Ther. (2012)
- Familial Wolff-Parkinson-White Syndrome: a disease of glycogen storage or ion channel dysfunction?. Light, P.E. J. Cardiovasc. Electrophysiol. (2006)
- Constitutively active adenosine monophosphate-activated protein kinase regulates voltage-gated sodium channels in ventricular myocytes. Light, P.E., Wallace, C.H., Dyck, J.R. Circulation (2003)
- Glucagon-like peptide-1 inhibits pancreatic ATP-sensitive potassium channels via a protein kinase A- and ADP-dependent mechanism. Light, P.E., Manning Fox, J.E., Riedel, M.J., Wheeler, M.B. Mol. Endocrinol. (2002)
- Distinct myoprotective roles of cardiac sarcolemmal and mitochondrial KATP channels during metabolic inhibition and recovery. Light, P.E., Kanji, H.D., Fox, J.E., French, R.J. FASEB J. (2001)
- Molecular basis of protein kinase C-induced activation of ATP-sensitive potassium channels. Light, P.E., Bladen, C., Winkfein, R.J., Walsh, M.P., French, R.J. Proc. Natl. Acad. Sci. U.S.A. (2000)
- Azimilide (Procter & Gamble). Light, P. IDrugs (2000)









