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Chemical Compound Review

naphthalene     naphthalene

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Disease relevance of naphthalene

  • To validate this notion in nonenteric bacteria, the expression and correct processing of LamB were confirmed by coupling the lamB gene to the salicylate-responsive Psa1 promoter of the NAH7 (naphthalene degradation) plasmid in Pseudomonas putida [1].

High impact information on naphthalene

  • The diffusion coefficient of NA inside the PRP-infinity particles (5.0+/-0.6) x 10(-11) cm2/s, agrees with the literature value that was obtained from the sorption rate of NA into the same particles [2].
  • The resulting peak-shaped IRF curve is used in two ways (i.e. subtraction and deconvolution) in order to correct the observed instantaneous rate curve of NA for the following experimental artifacts: hold-up volume of the bed and valve, transit-delay time between the bed and the detector and instrument bandbroadening of the NA zone [2].
  • The bronchiolar injury/repair response to naphthalene (NA) in mice includes acute distal airway epithelial injury that is followed by epithelial proliferation and redifferentiation, which result in repair of the epithelium within 14 days [3].
  • These results suggested that Pp60 is a protein that has an affinity for 2,4-D, NAA, and PCIB in vitro [4].
  • However, Naph-type metabolite contents increased significantly in liver cytosol at 4h exposure to 1% SF-STIUE, and at 24h exposure to 0.1% and 1% SF-STIUE [5].

Biological context of naphthalene

  • This constitutes virtually conclusive evidence for diffusion control of intra-particle kinetics of NA in the PS-DVB matrix of PRP-infinity and related polymers [2].

Associations of naphthalene with other chemical compounds

  • All of the examined transconjugants also catabolized phenanthrene, suggesting that Nah and Phn functions in U2, U28, and U31 strains are linked and probably encoded by transferable plasmids [6].

Gene context of naphthalene

  • Despite the low liver P450 content and EROD activity induction by NAPH and B(a)P, their genotoxic potential was higher than that of BNF [7].
  • Exposure reference values for BAP and NAPH-type metabolites could be used as a basis of comparison of exposure to PAH contamination [8].


  1. Nondisruptive detection of activity of catabolic promoters of Pseudomonas putida with an antigenic surface reporter system. Cebolla, A., Guzmán, C., de Lorenzo, V. Appl. Environ. Microbiol. (1996) [Pubmed]
  2. Continuous desorption rate measurement from a shallow-bed of poly(styrene-divinylbenzene) particles with correction for experimental artifacts. Bujalski, R., Cantwell, F.F. Journal of chromatography. A. (2004) [Pubmed]
  3. Prior exposure to aged and diluted sidestream cigarette smoke impairs bronchiolar injury and repair. Van Winkle, L.S., Evans, M.J., Brown, C.D., Willits, N.H., Pinkerton, K.E., Plopper, C.G. Toxicol. Sci. (2001) [Pubmed]
  4. Isolation and characterization of a 60 kDa 2,4-D-binding protein from the shoot apices of peach trees (Prunus persica L.); it is a homologue of protein disulfide isomerase. Sugaya, S., Ohmiya, A., Kikuchi, M., Hayashi, T. Plant Cell Physiol. (2000) [Pubmed]
  5. Dicentrarchus labrax biotransformation and genotoxicity responses after exposure to a secondary treated industrial/urban effluent. Gravato, C., Santos, M.A. Ecotoxicol. Environ. Saf. (2003) [Pubmed]
  6. Characterization of polycyclic aromatic hydrocarbons degradative soil Pseudomonas. Fuenmayor, S.L., Rodriguez Lemoine, V. Acta Cient. Venez. (1992) [Pubmed]
  7. Juvenile sea bass liver P450, EROD induction, and erythrocytic genotoxic responses to PAH and PAH-like compounds. Gravato, C., Santos, M.A. Ecotoxicol. Environ. Saf. (2002) [Pubmed]
  8. Reference values for exposure to PAH contaminants: Comparison of fish from Ohio and Mid-Atlantic streams. Cormier, S.M., Lin, E.L. Ecotoxicology (2006) [Pubmed]
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