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Gene Review

KY  -  kyphoscoliosis peptidase

Homo sapiens

Synonyms: FLJ33207, Kyphoscoliosis peptidase
 
 
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Disease relevance of KY

  • These results suggest that KY is an intrinsic part of the protein networks underlying the molecular mechanism of several limb-girdle muscular dystrophies, particularly those where interactions between filamin C and disease causing proteins have been shown [1].
  • Filamin C interacts with the muscular dystrophy KY protein and is abnormally distributed in mouse KY deficient muscle fibres [1].
 

High impact information on KY

  • An increase in the expression of stretch/stress response elements in fast and slow muscles has been previously described in a transcriptional profiling of KY deficient muscles [2].

References

  1. Filamin C interacts with the muscular dystrophy KY protein and is abnormally distributed in mouse KY deficient muscle fibres. Beatham, J., Romero, R., Townsend, S.K., Hacker, T., van der Ven, P.F., Blanco, G. Hum. Mol. Genet. (2004) [Pubmed]
  2. Constitutive upregulations of titin-based signalling proteins in KY deficient muscles. Beatham, J., Gehmlich, K., van der Ven, P.F., Sarparanta, J., Williams, D., Underhill, P., Geier, C., Fürst, D.O., Udd, B., Blanco, G. Neuromuscul. Disord. (2006) [Pubmed]
 
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