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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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incA  -  inclusion membrane protein A

Chlamydia trachomatis D/UW-3/CX

 
 
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Disease relevance of incA

  • Normal IncA expression and fusogenicity of inclusions in Chlamydia trachomatis isolates with the incA I47T mutation [1].
  • Chlamydial development is blocked in host cells transfected with Chlamydophila caviae incA [2].
 

High impact information on incA

  • Immediately downstream from the operon containing incD-G was the C. trachomatis homologue of incA [3].
  • In conclusion, the incA I47T mutation is not associated with the nonfusogenic phenotype [1].
  • Nucleotide-sequence analysis of the incA sequences from the variant isolates identified a variety of distinct sequence polymorphisms relative to incA from wild-type strains [4].
  • RESULTS: Cells transfected with plasmids expressing Chlamydophila caviae incA were not productively infected by C. caviae [2].
 

Analytical, diagnostic and therapeutic context of incA

References

  1. Normal IncA expression and fusogenicity of inclusions in Chlamydia trachomatis isolates with the incA I47T mutation. Pannekoek, Y., van der Ende, A., Eijk, P.P., van Marle, J., de Witte, M.A., Ossewaarde, J.M., van den Brule, A.J., Morré, S.A., Dankert, J. Infect. Immun. (2001) [Pubmed]
  2. Chlamydial development is blocked in host cells transfected with Chlamydophila caviae incA. Alzhanov, D., Barnes, J., Hruby, D.E., Rockey, D.D. BMC Microbiol. (2004) [Pubmed]
  3. Identification and characterization of a Chlamydia trachomatis early operon encoding four novel inclusion membrane proteins. Scidmore-Carlson, M.A., Shaw, E.I., Dooley, C.A., Fischer, E.R., Hackstadt, T. Mol. Microbiol. (1999) [Pubmed]
  4. Diversity within inc genes of clinical Chlamydia trachomatis variant isolates that occupy non-fusogenic inclusions. Rockey, D.D., Viratyosin, W., Bannantine, J.P., Suchland, R.J., Stamm, W.E. Microbiology (Reading, Engl.) (2002) [Pubmed]
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