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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
Gene Review

petH  -  ferredoxin-NADP oxidoreductase

Synechocystis sp. PCC 6803

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Disease relevance of petH

  • The phenotypic consequences of the genetic removal of this domain from the petH gene, which encodes FNR, have been studied in Synechocystis PCC 6803 [1].
  • A multiple gene family encodes FNR isoenzymes in plants, whereas there is only one FNR gene (petH) in cyanobacteria [2].
 

High impact information on petH

  • Site-directed mutants allowed us to conclude that the smaller isoform, FNR(S), is produced from an internal ribosome entry site within the petH ORF [2].
  • In the psaE mutant, the relative petH (encoding FNR) expression level was found to be significantly increased, providing a possible explanation for the lack of a phenotype (i.e., a decrease in growth rate) that was expected from the lower rate of linear electron transport in the mutant [3].
  • A model for a new type of bidirectional operator, regulating the expression of petH and prk, is proposed [4].

References

 
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