High impact information on Higher Nervous Activity

• Surprisingly, a diazo analog, (Z)-3-hexenyl diazoacetate, elicited slightly higher nervous activity than the natural ligand in the neurons specific and selective for (Z)-3-hexenyl acetate [1].
• The question of the formation of the two types of effect of vasopressin on higher nervous activity is discussed in relation to the evolution of mammals [2].
• The differences between the effects of ACTH and beta-endorphin on higher nervous activity are discussed, as are the possible mechanisms of these effects [3].
• The knowledge on the Common Brain Stem System (CBSS) described by Hess is reviewed in order to underline its role as a control centre integrating all the homeostatic and adaptive nervous activities, and in this context, the nervous control of the cardiovascular system is examined, particularly in relation to higher nervous activities [4].
• On acute effects of some drugs on the higher nervous activity in man. Ethanol (0.5 g/kg) and its interaction with diazepam (5 mg) and meclophenoxate (100 mg). Part LVIII [5].

Chemical compound and disease context of Higher Nervous Activity

• Acute effect of ethanol (0.3 g, 0.6 g and 0.9 g/kg) on the higher nervous activity in man [6].
• Effects of different drugs on the higher nervous activity in man. Meclophenoxate [7].
• Effects of different drugs on the higher nervous activity in man. Piracetam [8].
• The acute effect of diazepine derivatives on the higher nervous activity in man [9].
• On acute effects of some drugs on the higher nervous activity in man (the complex tactile analyser). Clorotepin (0.5 mg and 1.0 mg), methaqualone (300 mg) [10].

Gene context of Higher Nervous Activity

• On acute effects of some drugs on the higher nervous activity in man. Ethanol (0.1, 0.2, 0.4 g/kg). Part LVI [11].

References