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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
 
 

Immunohistochemical detection of the c-met proto-oncogene product in the congenital melanocytic nevus of an infant with neurocutaneous melanosis.

Transgenic mice overexpressing hepatocyte growth factor/scatter factor (HGF/ SF) demonstrate extensive pigmented nevi in both skin and leptomeninges of the central nervous system resembling human neurocutaneous melanosis. We immunohistochemically detected HGF/ SF receptor, Met, in a congenital nevus of an infant with neurocutaneous melanosis, indicating that deregulation of HGF/ SF-Met signaling in the critical period of development may lead to this fatal syndrome.[1]

References

  1. Immunohistochemical detection of the c-met proto-oncogene product in the congenital melanocytic nevus of an infant with neurocutaneous melanosis. Takayama, H., Nagashima, Y., Hara, M., Takagi, H., Mori, M., Merlino, G., Nakazato, Y. J. Am. Acad. Dermatol. (2001) [Pubmed]
 
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