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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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CYLD in ubiquitin signaling and tumor pathogenesis.

Absence of CYLD, which encodes a deubiquitinating enzyme, causes an inherited disease characterized by benign skin tumors. In this issue of Cell, Massoumi et al. (2006) show that CYLD deubiquitinates the coactivator Bcl-3, thereby preventing its translocation into the nucleus, where it normally interacts with NF-kappaB and activates transcription of proliferation genes in response to growth signals.[1]

References

  1. CYLD in ubiquitin signaling and tumor pathogenesis. Ikeda, F., Dikic, I. Cell (2006) [Pubmed]
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