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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
 
 

uvrB-dependent, recF-independent post-replication (or replication) repair in Escherichia coli.

In UV-damaged cells, a large fraction of pyrimidine dimers may remain unexcised and may be tolerated by a uvrB recA lexA-dependent non-excisional mode of repair (M. Sedliaková, J. Brozmanová, F. Maŝek and K. Kleibl, Biophys. J., 36 (1981) 429-441). We show here that a similar repair pathway operates in the Escherichia coli recF 143 single mutant but not in the recF uvrB double mutant. This indicates that the putative repair pathway is recF independent.[1]

References

  1. uvrB-dependent, recF-independent post-replication (or replication) repair in Escherichia coli. Slezáriková, V., Sedliaková, M. J. Photochem. Photobiol. B, Biol. (1991) [Pubmed]
 
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