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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Flaxseed and its mammalian lignan precursor cause a lengthening or cessation of estrous cycling in rats.

Flaxseed and its mammalian lignan precursor secoisolariciresinol diglycoside (SDG) have been shown to be mammary cancer-protective in rats. Thus, the antiestrogenic effects of flaxseed and SDG were compared with tamoxifen, an antiestrogen, by monitoring rat estrous cycling. Four-week supplementation of a high-fat diet with flaxseed (2.5, 5, or 10%) or SDG (0.75, 1.5 or 3.0 mg/day) produced a dose-related cessation or lengthening (by 18-39%) of estrous cycles in up to 66% of rats. With tamoxifen (1 mg/kg body weight/day), 83% of the animals had irregular cycles or were in persistent diestrus. Flaxseed and SDG were antiestrogenic without gross tissue toxicity.[1]

References

  1. Flaxseed and its mammalian lignan precursor cause a lengthening or cessation of estrous cycling in rats. Orcheson, L.J., Rickard, S.E., Seidl, M.M., Thompson, L.U. Cancer Lett. (1998) [Pubmed]
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