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Ibrahim, A.S. et al.

Ibrahim, Filler, Sanglard, Edwards, Hube,

Secreted aspartyl proteinases and interactions of Candida albicans with human endothelial cells.

The endothelial cell interactions of homozygous null mutants of Candida albicans that were deficient in secreted aspartyl proteinase 1 ( Sap1), Sap2, or Sap3 were investigated. Only Sap2 was found to contribute to the ability of C. albicans to damage endothelial cells and stimulate them to express E-selectin. None of the Saps studied appears to play a role in C. albicans adherence to endothelial cells.[1]

References

Secreted aspartyl proteinases and interactions of Candida albicans with human endothelial cells. Ibrahim, A.S., Filler, S.G., Sanglard, D., Edwards, J.E., Hube, B. Infect. Immun. (1998) [Pubmed]

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