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Rohit N. Kulkarni

Islet Cell Biology and Regenerative Medicine

Joslin Diabetes Center

Harvard Medical School

Boston

USA

[email]@*.harvard.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Islet Cell Biology and Regenerative Medicine, Joslin Diabetes Center, Harvard Medical School, Boston, USA. 2002 - 2012
  • 602 One Joslin Place, Boston Massachusetts 02215. 2012
  • Joslin Diabetes Center, One Joslin Place, Room 602, Boston, USA. 2003 - 2004

References

  1. Human β-cell proliferation and intracellular signaling: driving in the dark without a road map. Kulkarni, R.N., Mizrachi, E.B., Ocana, A.G., Stewart, A.F. Diabetes (2012) [Pubmed]
  2. Enhanced GLP-1- and Sulfonylurea-Induced Insulin Secretion in Islets Lacking Leptin Signaling. Morioka, T., Dishinger, J.F., Reid, K.R., Liew, C.W., Zhang, T., Inaba, M., Kennedy, R.T., Kulkarni, R.N. Mol. Endocrinol. (2012) [Pubmed]
  3. GIP: no longer the neglected incretin twin? Kulkarni, R.N. Sci. Transl. Med (2010) [Pubmed]
  4. Uncoupling modifier genes from uncoupling protein 2 in pancreatic beta-cells. Kulkarni, R.N. Endocrinology (2009) [Pubmed]
  5. Ephs and ephrins keep pancreatic Beta cells connected. Kulkarni, R.N., Kahn, C.R. Cell (2007) [Pubmed]
  6. New insights into the roles of insulin/IGF-I in the development and maintenance of beta-cell mass. Kulkarni, R.N. Rev. Endocr. Metab. Disord (2005) [Pubmed]
  7. The islet beta-cell. Kulkarni, R.N. Int. J. Biochem. Cell Biol. (2004) [Pubmed]
  8. Molecular biology. HNFs--linking the liver and pancreatic islets in diabetes. Kulkarni, R.N., Kahn, C.R. Science (2004) [Pubmed]
  9. Islet secretory defect in insulin receptor substrate 1 null mice is linked with reduced calcium signaling and expression of sarco(endo)plasmic reticulum Ca2+-ATPase (SERCA)-2b and -3. Kulkarni, R.N., Roper, M.G., Dahlgren, G., Shih, D.Q., Kauri, L.M., Peters, J.L., Stoffel, M., Kennedy, R.T. Diabetes (2004) [Pubmed]
  10. Lessons for human diabetes from experimental mouse models. Kulkarni, R.N., Zisman, A. Curr. Diab. Rep. (2003) [Pubmed]
  11. beta-cell-specific deletion of the Igf1 receptor leads to hyperinsulinemia and glucose intolerance but does not alter beta-cell mass. Kulkarni, R.N., Holzenberger, M., Shih, D.Q., Ozcan, U., Stoffel, M., Magnuson, M.A., Kahn, C.R. Nat. Genet. (2002) [Pubmed]
  12. Receptors for insulin and insulin-like growth factor-1 and insulin receptor substrate-1 mediate pathways that regulate islet function. Kulkarni, R.N. Biochem. Soc. Trans. (2002) [Pubmed]
  13. Tissue-specific targeting of the insulin receptor gene. Kulkarni, R.N., Okada, T. Endocrine (2002) [Pubmed]
 
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