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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Steffen Rossner

Paul Flechsig Institute for Brain Research

Department of Neurochemistry

University of Leipzig

Jahnallee 59

Germany

[email]@*.uni-leipzig.de

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Paul Flechsig Institute for Brain Research, Department of Neurochemistry, University of Leipzig, Jahnallee 59, Germany. 2000 - 2006
  • Department of Neurochemistry, Paul Flechsig Institute for Brain Research, Leipzig, Germany. 1999 - 2005

References

  1. Transcriptional and translational regulation of BACE1 expression--implications for Alzheimer's disease. Rossner, S., Sastre, M., Bourne, K., Lichtenthaler, S.F. Prog. Neurobiol. (2006) [Pubmed]
  2. Alzheimer's disease beta-secretase BACE1 is not a neuron-specific enzyme. Rossner, S., Lange-Dohna, C., Zeitschel, U., Perez-Polo, J.R. J. Neurochem. (2005) [Pubmed]
  3. Brain prolyl endopeptidase expression in aging, APP transgenic mice and Alzheimer's disease. Rossner, S., Schulz, I., Zeitschel, U., Schliebs, R., Bigl, V., Demuth, H.U. Neurochem. Res. (2005) [Pubmed]
  4. Munc13-1-mediated vesicle priming contributes to secretory amyloid precursor protein processing. Rossner, S., Fuchsbrunner, K., Lange-Dohna, C., Hartlage-Rübsamen, M., Bigl, V., Betz, A., Reim, K., Brose, N. J. Biol. Chem. (2004) [Pubmed]
  5. New players in old amyloid precursor protein-processing pathways. Rossner, S. Int. J. Dev. Neurosci. (2004) [Pubmed]
  6. Increased neuronal and glial expression of protein kinase C isoforms in neocortex of transgenic Tg2576 mice with amyloid pathology. Rossner, S., Mehlhorn, G., Schliebs, R., Bigl, V. Eur. J. Neurosci. (2001) [Pubmed]
  7. Protein kinase Calpha and beta1 isoforms are regulators of alpha-secretory proteolytic processing of amyloid precursor protein in vivo. Rossner, S., Mendla, K., Schliebs, R., Bigl, V. Eur. J. Neurosci. (2001) [Pubmed]
  8. Neuronal and glial beta-secretase (BACE) protein expression in transgenic Tg2576 mice with amyloid plaque pathology. Rossner, S., Apelt, J., Schliebs, R., Perez-Polo, J.R., Bigl, V. J. Neurosci. Res. (2001) [Pubmed]
  9. Intracerebroventricular infusion of CHO5, a rat monoclonal antibody directed against mouse low-affinity nerve growth factor receptor (p75NTR), specifically labels basal forebrain cholinergic neurons in mouse brain. Rossner, S., Schliebs, R., Bigl, V. Metab. Brain. Dis (2000) [Pubmed]
  10. Constitutive overactivation of protein kinase C in guinea pig brain increases alpha-secretory APP processing without decreasing beta-amyloid generation. Rossner, S., Beck, M., Stahl, T., Mendla, K., Schliebs, R., Bigl, V. Eur. J. Neurosci. (2000) [Pubmed]
  11. Regulated secretion of amyloid precursor protein by TrkA receptor stimulation in rat pheochromocytoma-12 cells is mitogen activated protein kinase sensitive. Rossner, S., Ueberham, U., Schliebs, R., Perez-Polo, J.R., Bigl, V. Neurosci. Lett. (1999) [Pubmed]
 
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